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Nelli Mnatsakanyan

Pennsylvania State University · US
Area of research
Molecular Biology · Electrical and Electronic Engineering
Research interest
Research focused on ATP synthase and Protein subunit, with related work in Mitochondrion, Mitochondrial permeability transition pore, Cell biology. Notable publications include 'Identity, structure, and function of the mitochondrial permeability transition pore: controversies, consensus, recent advances, and future directions', 'ATP Synthase C-Subunit-Deficient Mitochondria Have a Small Cyclosporine A-Sensitive Channel, but Lack the Permeability Transition Pore', and 'ATP Synthase c-Subunit Leak Causes Aberrant Cellular Metabolism in Fragile X Syndrome'.
h-index
citations
868
works
11
NIH funding
primary concept
email

Recent publications

Bedaquiline inhibits the ATP synthase leak channel and prevents glutamate-induced neuronal death
bioRxiv (Cold Spring Harbor Laboratory) 2026cited by 1position: lastdoi
Identity, structure, and function of the mitochondrial permeability transition pore: controversies, consensus, recent advances, and future directions
Cell Death and Differentiation 2023cited by 371position: middledoi
Mitochondrial ATP synthase c-subunit leak channel triggers cell death upon loss of its F1 subcomplex
Cell Death and Differentiation 2022cited by 48position: firstdoi
Fluid shear stress enhances proliferation of breast cancer cells via downregulation of the c-subunit of the F1FO ATP synthase
Biochemical and Biophysical Research Communications 2022cited by 9position: middledoi
ATP Synthase c-Subunit Leak Causes Aberrant Cellular Metabolism in Fragile X Syndrome
Cell 2020cited by 116position: middledoi
ATP Synthase C-Subunit-Deficient Mitochondria Have a Small Cyclosporine A-Sensitive Channel, but Lack the Permeability Transition Pore
Cell Reports 2019cited by 181position: middledoi
Parkinson’s disease protein DJ-1 regulates ATP synthase protein components to increase neuronal process outgrowth
Cell Death and Disease 2019cited by 95position: middledoi
Alpha-Tocotrienol Prevents Oxidative Stress-Mediated Post-Translational Cleavage of Bcl-xL in Primary Hippocampal Neurons
International Journal of Molecular Sciences 2019cited by 22position: middledoi
Mitochondrial Megachannel Resides in Monomeric ATP Synthase
Biophysical Journal 2019cited by 2position: firstdoi
Vitamin E Prevents ΔN-Bcl-xL-associate Mitochondrial Dysfunction in Primary Hippocampal Neurons (P14-024-19)
Current Developments in Nutrition 2019cited by 1position: middledoi
Functional Chimeras of GLIC Obtained by Adding the Intracellular Domain of Anion- and Cation-Conducting Cys-Loop Receptors
Biochemistry 2015cited by 22position: firstdoi

Grants

No grants ingested yet.

Frequent collaborators

Elizabeth A. Jonas · Yale University9 papers (2019–2023)Hana Park · Seoul National University7 papers (2019–2022)Paige Miranda · New York University4 papers (2019–2022)Pawel Licznerski · Yale University3 papers (2019–2020)Morven Graham · Yale University3 papers (2019–2022)Evgeny V. Pavlov · New York University3 papers (2019–2023) · 2 papers (2019–2019)Besnik Murtishi · Pennsylvania State University2 papers (2019–2022)Jing Wu · Pennsylvania State University2 papers (2019–2022) · 2 papers (2019–2020)Rongmin Chen · Florida State University2 papers (2019–2020)Marc C. Llaguno · Southwestern Medical Center2 papers (2019–2022)Richard J. Levy · Columbia University Irving Medical Center2 papers (2020–2022) · 1 papers (2019–2019) · 1 papers (2020–2020) · 1 papers (2020–2020)Shey‐Shing Sheu · University of Rochester1 papers (2023–2023) · 1 papers (2019–2019)Kambiz N. Alavian · Imperial College Healthcare NHS Trust1 papers (2019–2019) · 1 papers (2020–2020)