Area of research
Immunology · Rehabilitation
Research interest
Research interests include Immune Cell Function and Interaction, T-cell and B-cell Immunology, Wound Healing and Treatments, and Immunotherapy and Immune Responses.
Selective requirement of glycosphingolipid synthesis for natural killer and cytotoxic T cells
Paxbp1 restrains cytotoxic and innate cell programs in CD4+ T cells to promote Th2 differentiation
SOX2-Dependent Wound Repair Signature Triggers Prohealing Outcome in Hyperglycemic Wounds
Inflammation switches the chemoattractant requirements for naive lymphocyte entry into lymph nodes
Reprogramming of epidermal keratinocytes by PITX1 transforms the cutaneous cellular landscape and promotes wound healing.
Identification of Fibroinflammatory and Fibrotic Transcriptomic Subsets of Human Cutaneous Sclerotic Chronic Graft-Versus-Host Disease
Constitutively active Lyn kinase causes a cutaneous small vessel vasculitis and liver fibrosis syndrome.
Spleen tyrosine kinase inhibition restores myeloid homeostasis in COVID-19.
Divergent roles for STAT4 in shaping differentiation of cytotoxic ILC1 and NK cells during gut inflammation
Divergent roles for STAT4 in shaping differentiation of cytotoxic ILC1 and NK cells during gut inflammation.
Chromatin Landscape Governing Murine Epidermal Differentiation
Microbial ligand-independent regulation of lymphopoiesis by NOD1.
A central role for STAT5 in the transcriptional programing of T helper cell metabolism
A central role for STAT5 in the transcriptional programing of T helper cell metabolism.
Genetically programmed alternative splicing of NEMO mediates an autoinflammatory disease phenotype.
Identification of fibroinflammatory and fibrotic transcriptomic subsets of human sclerotic cutaneous chronic graft-versus-host disease
Somatic Mutations in <i>UBA1</i> and Severe Adult-Onset Autoinflammatory Disease
Deregulated immune cell recruitment orchestrated by FOXM1 impairs human diabetic wound healing
Distinct interferon signatures and cytokine patterns define additional systemic autoinflammatory diseases.
FoxO maintains a genuine muscle stem-cell quiescent state until geriatric age
Neutrophil extracellular traps mediate articular cartilage damage and enhance cartilage component immunogenicity in rheumatoid arthritis.
Distinct interferon signatures and cytokine patterns define additional systemic autoinflammatory diseases
Gata6+ Pericardial Cavity Macrophages Relocate to the Injured Heart and Prevent Cardiac Fibrosis
The Magnitude of IFN-γ Responses Is Fine-Tuned by DNA Architecture and the Non-coding Transcript of Ifng-as1
SOX2 Epidermal Overexpression Promotes Cutaneous Wound Healing via Activation of EGFR/MEK/ERK Signaling Mediated by EGFR Ligands
SOX2 Epidermal Overexpression Promotes Cutaneous Wound Healing via Activation of EGFR/MEK/ERK Signaling Mediated by EGFR Ligands.
183 A phase 1B/2A trial of tofacitinib, an oral janus kinase inhibitor, in systemic lupus erythematosus
JAK1/2 inhibition with baricitinib in the treatment of autoinflammatory interferonopathies
Transcriptional signature primes human oral mucosa for rapid wound healing
Activated STING in a Vascular and Pulmonary Syndrome