Area of research
Physiology · Psychiatry and Mental health
Research interest
Research focused on Neuroscience and Chronic pain, with related work in Translocator protein, Fibromyalgia, Default mode network. Notable publications include 'Evidence for brain glial activation in chronic pain patients', 'Default mode network connectivity encodes clinical pain: An arterial spin labeling study', and 'Brain glial activation in fibromyalgia – A multi-site positron emission tomography investigation'.
ENIGMA-Chronic Pain: a worldwide initiative to identify brain correlates of chronic pain
Extra-axial inflammatory signal and its relationship to peripheral and central immunity in depression
Characterization of cortico-meningeal translocator protein expression in multiple sclerosis
Physically informed deep neural networks for metabolite-corrected plasma input function estimation in dynamic PET imaging
The Back Pain Consortium (BACPAC) Research Program: Structure, Research Priorities, and Methods
The pandemic brain: Neuroinflammation in non-infected individuals during the COVID-19 pandemic
Boston biorepository, recruitment and integrative network (BBRAIN): A resource for the Gulf War Illness scientific community
The Neuroimmunology of Chronic Pain: From Rodents to Humans
In-vivo imaging of neuroinflammation in veterans with Gulf War illness
Brain glial activation in fibromyalgia – A multi-site positron emission tomography investigation
Neuroinflammation in Huntington’s Disease: New Insights with <sup>11</sup>C-PBR28 PET/MRI
Pharmacological Modulation of Noradrenergic Arousal Circuitry Disrupts Functional Connectivity of the Locus Ceruleus in Humans
Pseudoreference Regions for Glial Imaging with <sup>11</sup>C-PBR28: Investigation in 2 Clinical Cohorts
Evidence for brain glial activation in chronic pain patients
The Somatosensory Link in Fibromyalgia: Functional Connectivity of the Primary Somatosensory Cortex Is Altered by Sustained Pain and Is Associated With Clinical/Autonomic Dysfunction
The Lateral Prefrontal Cortex Mediates the Hyperalgesic Effects of Negative Cognitions in Chronic Pain Patients
Disruption of thalamic functional connectivity is a neural correlate of dexmedetomidine-induced unconsciousness
Default mode network connectivity encodes clinical pain: An arterial spin labeling study