Area of research
Cellular and Molecular Neuroscience · Cognitive Neuroscience
Research interest
Research focused on Neuroscience and Lesion, with related work in Long-term potentiation, Diaschisis, Ectodomain. Notable publications include 'Oligodendrocyte Precursor Cells Modulate the Neuronal Network by Activity-Dependent Ectodomain Cleavage of Glial NG2', 'BDNF contributes to the facilitation of hippocampal synaptic plasticity and learning enabled by environmental enrichment', and 'Microglial A20 Protects the Brain from CD8 T-Cell-Mediated Immunopathology'.
Postnatal development of vasoactive intestinal polypeptide‐expressing <scp>GABAergic</scp> interneurons in mouse somatosensory cortex
Temporal characterisation and electrophysiological implications of TBI-induced serine/threonine kinase activity in mouse cortex
Functional Alterations in Parvalbumin-Positive Interneurons after Traumatic Brain Injury in Somatosensory Cortex of Mice
Lysosomal TRPML1 activation modulates synaptic transmission and intrinsic neuronal excitability
Tonic activation of GABAB receptors via GAT-3 mediated GABA release reduces network activity in the developing somatosensory cortex in GAD67-GFP mice
The K63 deubiquitinase CYLD modulates autism-like behaviors and hippocampal plasticity by regulating autophagy and mTOR signaling
Early life adversity targets the transcriptional signature of hippocampal NG2+ glia and affects voltage gated sodium (Nav) channels properties
Adaptive Mechanisms of Somatostatin-Positive Interneurons after Traumatic Brain Injury through a Switch of α Subunits in L-Type Voltage-Gated Calcium Channels
Nitric Oxide/Cyclic Guanosine Monophosphate Signaling via Guanylyl Cyclase Isoform 1 Mediates Early Changes in Synaptic Transmission and Brain Edema Formation after Traumatic Brain Injury
Microglial A20 Protects the Brain from CD8 T-Cell-Mediated Immunopathology
Proteasome and Autophagy-Mediated Impairment of Late Long-Term Potentiation (l-LTP) after Traumatic Brain Injury in the Somatosensory Cortex of Mice
Maladaptive cortical hyperactivity upon recovery from experimental autoimmune encephalomyelitis
Neuronal excitability and spontaneous synaptic transmission in the entorhinal cortex of BDNF heterozygous mice
Nitric oxide/cGMP signaling via guanylyl cyclase isoform 1 modulates glutamate and GABA release in somatosensory cortex of mice
Acute Cortical Transhemispheric Diaschisis after Unilateral Traumatic Brain Injury
Physiological Properties of Supragranular Cortical Inhibitory Interneurons Expressing Retrograde Persistent Firing
Oligodendrocyte Precursor Cells Modulate the Neuronal Network by Activity-Dependent Ectodomain Cleavage of Glial NG2
BDNF contributes to the facilitation of hippocampal synaptic plasticity and learning enabled by environmental enrichment
Neuronal mechanisms underlying transhemispheric diaschisis following focal cortical injuries
NO regulates the strength of synaptic inputs onto hippocampal CA1 neurons via NO-GC1/cGMP signalling
Postsynaptic NO/cGMP Increases NMDA Receptor Currents via Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels in the Hippocampus
The laser lesion of the mouse visual cortex as a model to study neural extracellular matrix remodeling during degeneration, regeneration and plasticity of the CNS
Shift from phasic to tonic GABAergic transmission following laser-lesions in the rat visual cortex