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Gustaf Wigerblad

National Institutes of Health · US
Area of research
Immunology · Rheumatology
Research interest
Research interests include Neutrophil, Myeloperoxidase and Oxidative Mechanisms, Cell Adhesion Molecules Research, Rheumatoid Arthritis Research and Therapies, and Systemic Lupus Erythematosus Research.
h-index
23
citations
4,542
works
43
NIH funding
primary concept
Medicine
email

Recent publications

BTK drives neutrophil activation for sterilizing antifungal immunity
Journal of Clinical Investigation 2024cited by 27position: middledoi
Somatic Mutations in <i>UBA1</i> and Severe Adult-Onset Autoinflammatory Disease
New England Journal of Medicine 2020cited by 1,122position: middledoi
Cartilage-binding antibodies induce pain through immune complex–mediated activation of neurons
The Journal of Experimental Medicine 2019cited by 102position: middledoi
Differential ACPA Binding to Nuclear Antigens Reveals a PAD-Independent Pathway and a Distinct Subset of Acetylation Cross-Reactive Autoantibodies in Rheumatoid Arthritis
Frontiers in Immunology 2019cited by 59position: middledoi
Pathogenic Citrulline‐Multispecific B Cell Receptor Clades in Rheumatoid Arthritis
Arthritis & Rheumatology 2018cited by 111position: middledoi
Inflammation-induced GluA1 trafficking and membrane insertion of Ca2+ permeable AMPA receptors in dorsal horn neurons is dependent on spinal tumor necrosis factor, PI3 kinase and protein kinase A
Experimental Neurology 2017cited by 35position: firstdoi
Pain in Rheumatoid Arthritis: Models and Mechanisms
Pain Management 2016cited by 75position: middledoi
Identification of a novel chemokine-dependent molecular mechanism underlying rheumatoid arthritis-associated autoantibody-mediated bone loss
Annals of the Rheumatic Diseases 2015cited by 339position: middledoi
Autoantibodies to citrullinated proteins induce joint pain independent of inflammation via a chemokine-dependent mechanism
Annals of the Rheumatic Diseases 2015cited by 252position: firstdoi
Olfactory exposure to males, including men, causes stress and related analgesia in rodents
Nature Methods 2014cited by 937position: middledoi
Spinal Actions of Lipoxin A4 and 17(R)-Resolvin D1 Attenuate Inflammation-Induced Mechanical Hypersensitivity and Spinal TNF Release
PLoS ONE 2013cited by 72position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Camilla I. Svensson · Karolinska Institutet7 papers (2013–2018)Vivianne Malmström · Karolinska Institutet4 papers (2015–2019)Lars Klareskog · Karolinska University Hospital4 papers (2015–2019)Khaled Amara · Karolinska University Hospital3 papers (2015–2018)Duygu B. Bas · Karolinska Institutet3 papers (2013–2016)Jungo Kato · University of Tsukuba2 papers (2015–2015)Philip J. Titcombe · University of Minnesota Medical Center2 papers (2018–2019)Per Johan Jakobsson · Karolinska University Hospital2 papers (2015–2015)Juan Miguel Jiménez‐Andrade · University of Arizona2 papers (2015–2015)Cátia Fernandes‐Cerqueira · Karolinska University Hospital2 papers (2015–2015)Jie Su · Karolinska Institutet2 papers (2015–2016)Akilan Krishnamurthy · American College of Rheumatology2 papers (2015–2015)Karl Skriner · Freie Universität Berlin2 papers (2018–2019)Anca I. Catrina · Karolinska University Hospital2 papers (2015–2015)Peter Ström · University of Pittsburgh2 papers (2018–2019)Karin Lundberg · Karolinska University Hospital2 papers (2015–2015)Daniel L. Mueller · University of Minnesota, Twin Cities2 papers (2018–2019)Timothy B. Niewold · Hospital for Special Surgery1 papers (2018–2018)Alan Schumann · University of Alabama at Birmingham1 papers (2014–2014)Johan Rönnelid · Science for Life Laboratory1 papers (2019–2019)