Area of research
Genetics · Oncology
Research interest
Research interests include Digestive system and related health, Inflammatory Bowel Disease, Helicobacter pylori-related gastroenterology studies, and Cancer Cells and Metastasis.
Generating vasculature and immune cell-chimeric tumoroids via intraperitoneal Xenograft
Clinical utility of hematological parameters in aplastic anemia
The RNA N6-methyladenosine methylome coordinates long non-coding RNAs to mediate cancer drug resistance by activating PI3K signaling
Corrigendum to "Monogenic deficiency in murine intestinal Cdc42 leads to mucosal inflammation that induces crypt dysplasia" [Genes & Dis 11 (2024) 413-429].
Innovative nebulization delivery of lipid nanoparticle-encapsulated siRNA: a therapeutic advance for Staphylococcus aureus-induced pneumonia
Monogenic deficiency in murine intestinal Cdc42 leads to mucosal inflammation that induces crypt dysplasia.
Interaction of the intestinal cytokines-JAKs-STAT3 and 5 axes with RNA N6-methyladenosine to promote chronic inflammation-induced colorectal cancer.
Lemierre's syndrome complicating multiple organ failure caused by Fusobacterium necrophorum subsp. funduliforme F1260: Case report and review
Clinical and molecular characterization of early onset intrahepatic cholangiocarcinoma
996P Transarterial chemoembolization combined donafenib with/without programmed death-1 inhibitors for initially unresectable hepatocellular carcinoma in a multicenter retrospective study
A centric view of JAK/STAT5 in intestinal homeostasis, infection, and inflammation.
Protein O-GlcNAc Modification Links Dietary and Gut Microbial Cues to the Differentiation of Enteroendocrine L Cells.
Hepatic growth hormone - JAK2 - STAT5 signalling: Metabolic function, non-alcoholic fatty liver disease and hepatocellular carcinoma progression.
Constitutive STAT5 activation regulates Paneth and Paneth-like cells to control <i>Clostridium difficile</i> colitis.
Cell Metabolism Control Through O-GlcNAcylation of STAT5: A Full or Empty Fuel Tank Makes a Big Difference for Cancer Cell Growth and Survival.
Lgr5<sup>+</sup> intestinal stem cell sorting and organoid culture.
Deficiency in intestinal epithelial O‐GlcNAcylation predisposes to gut inflammation
BIN1 reverses PD-L1-mediated immune escape by inactivating the c-MYC and EGFR/MAPK signaling pathways in non-small cell lung cancer
O-GlcNAcylation of STAT5 controls tyrosine phosphorylation and oncogenic transcription in STAT5-dependent malignancies
Methylation decreases the Bin1 tumor suppressor in ESCC and restoration by decitabine inhibits the epithelial mesenchymal transition
Potent therapeutic target of inflammation, virus and tumor: focus on interleukin-27
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