Area of research
Molecular Biology · Virology
Research interest
Research interests include HIV Research and Treatment, CRISPR and Genetic Engineering, Virus-based gene therapy research, and Genomics and Chromatin Dynamics.
Tissue-specific restriction of transposon-derived regulatory elements safeguards cell-type identity
An overexpression platform reveals the functional diversity of human KRAB-Zinc Finger Proteins in maintaining cellular homeostasis
Antigenic landscape of a highly mutated SARS-CoV-2 Spike in ongoing viral evolution
Cancer cells subvert the primate-specific KRAB zinc finger protein ZNF93 to control APOBEC3B.
Zinc-finger proteins with a co-opted capsid domain anchor nucleosomes over transposon sequences
Transposable element co-option drives transcription factor neofunctionalization
Tissue-specific restriction of TE-derived regulatory elements safeguards cell-type identity
Identification of a lipid oxygen radical defense pathway and its epigenetic control.
Identification of the
Lipid Oxygen Radical Defense pathway and its epigenetic control
Identification of a Lipid Oxygen Radical Defense pathway and its epigenetic control
A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress-Induced Inflammation.
Clusters of lineage-specific genes are anchored by ZNF274 in repressive perinucleolar compartments.
Ancestral genome reconstruction enhances transposable element annotation by identifying degenerate integrants
Transposable elements mediate genetic effects altering the expression of nearby genes in colorectal cancer.
Evolutionarily recent transcription factors partake in human cell cycle regulation
Figure S2 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Figure S2 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Figure S5 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Clusters of lineage-specific genes are anchored by ZNF274 in repressive perinucleolar compartments
Data from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Figure S1 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Figure S6 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Figure S5 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Data from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Figure S3 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Figure S1 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Figure S4 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Figure S3 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Figure S4 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation
Figure S6 from A Cluster of Evolutionarily Recent KRAB Zinc Finger Proteins Protects Cancer Cells from Replicative Stress–Induced Inflammation