Akrit Sodhi

Hypoxia-induced BNIP3 facilitates the progression and metastasis of uveal melanoma by driving metabolic reprogramming

The AKT2/SIRT5/TFEB pathway as a potential therapeutic target in non-neovascular AMD

Targeting hypoxia-inducible factors with 32-134D safely and effectively treats diabetic eye disease in mice

PAI-1 is a vascular cell–specific HIF-2–dependent angiogenic factor that promotes retinal neovascularization in diabetic patients

HIF-1α and HIF-2α redundantly promote retinal neovascularization in patients with ischemic retinal disease

Glycolysis links reciprocal activation of myeloid cells and endothelial cells in the retinal angiogenic niche

Inhibition of soluble epoxide hydrolase prevents diabetic retinopathy

Endothelial adenosine A2a receptor-mediated glycolysis is essential for pathological retinal angiogenesis

Hypoxia-inducible factor 1 upregulation of both VEGF and ANGPTL4 is required to promote the angiogenic phenotype in uveal melanoma

Angiopoietin-like 4 is a potent angiogenic factor and a novel therapeutic target for patients with proliferative diabetic retinopathy

Hippo-Independent Activation of YAP by the GNAQ Uveal Melanoma Oncogene through a Trio-Regulated Rho GTPase Signaling Circuitry

Hypoxic retinal Müller cells promote vascular permeability by HIF-1–dependent up-regulation of angiopoietin-like 4

VEGF Secreted by Hypoxic Müller Cells Induces MMP-2 Expression and Activity in Endothelial Cells to Promote Retinal Neovascularization in Proliferative Diabetic Retinopathy