Area of research
Infectious Diseases · Molecular Biology
Research interest
Research interests include Antimicrobial Resistance in Staphylococcus, Biochemical and Structural Characterization, Vector-borne infectious diseases, and Viral Infections and Vectors.
Glycosylation-dependent opsonophagocytic activity of staphylococcal protein A antibodies
FPR1 is the plague receptor on host immune cells
<i>Rickettsia conorii</i> O antigen is the target of bactericidal Weil–Felix antibodies
Antibodies against a secreted product of <i>Staphylococcus aureus</i> trigger phagocytic killing
Peptidoglycan-linked protein A promotes T cell-dependent antibody expansion during <i>Staphylococcus aureus</i> infection
EssE Promotes Staphylococcus aureus ESS-Dependent Protein Secretion To Modify Host Immune Responses during Infection
Pathogenic conversion of coagulase-negative staphylococci
Staphylococcal manipulation of host immune responses
Protein A Suppresses Immune Responses during Staphylococcus aureus Bloodstream Infection in Guinea Pigs
Mouse models for infectious diseases caused by Staphylococcus aureus
<i>Staphylococcus aureus</i> infection induces protein A–mediated immune evasion in humans
The Capsular Polysaccharide of Staphylococcus aureus Is Attached to Peptidoglycan by the LytR-CpsA-Psr (LCP) Family of Enzymes
Protein A-neutralizing monoclonal antibody protects neonatal mice against Staphylococcus aureus
Role of Protein A in the Evasion of Host Adaptive Immune Responses by Staphylococcus aureus
Small molecule inhibitor of lipoteichoic acid synthesis is an antibiotic for Gram-positive bacteria
N-Acetylglucosaminylation of Serine-Aspartate Repeat Proteins Promotes Staphylococcus aureus Bloodstream Infection
ADAM10 Mediates Vascular Injury Induced by Staphylococcus aureus α-Hemolysin
Protein A-Specific Monoclonal Antibodies and Prevention of Staphylococcus aureus Disease in Mice
Abscess Formation and Alpha-Hemolysin Induced Toxicity in a Mouse Model of Staphylococcus aureus Peritoneal Infection