Area of research
Neurology · Molecular Biology
Research interest
Research interests include Vagus Nerve Stimulation Research, Nicotinic Acetylcholine Receptors Study, Advanced Glycation End Products research, and Neuroinflammation and Neurodegeneration Mechanisms.
Stimulation of the hepatoportal nerve plexus with focused ultrasound restores glucose homoeostasis in diabetic mice, rats and swine
High-frequency electrical stimulation attenuates neuronal release of inflammatory mediators and ameliorates neuropathic pain
HMGB1 released from nociceptors mediates inflammation
The Fourth Bioelectronic Medicine Summit “Technology Targeting Molecular Mechanisms”: current progress, challenges, and charting the future
Identification of a brainstem locus that inhibits tumor necrosis factor
The microbiota regulate neuronal function and fear extinction learning
Inhibition of HMGB1/RAGE-mediated endocytosis by HMGB1 antagonist box A, anti-HMGB1 antibodies, and cholinergic agonists suppresses inflammation
Forebrain Cholinergic Signaling Regulates Innate Immune Responses and Inflammation
Galantamine alleviates inflammation and insulin resistance in patients with metabolic syndrome in a randomized trial
A novel flexible cuff-like microelectrode for dual purpose, acute and chronic electrical interfacing with the mouse cervical vagus nerve
Identification of CD163 as an antiinflammatory receptor for HMGB1-haptoglobin complexes
Blood pressure regulation by CD4+ lymphocytes expressing choline acetyltransferase
Neuronal Circuits Modulate Antigen Flow Through Lymph Nodes
MD-2 is required for disulfide HMGB1–dependent TLR4 signaling
High Mobility Group Box Protein 1 (HMGB1): The Prototypical Endogenous Danger Molecule
Single-Pulse and Unidirectional Electrical Activation of the Cervical Vagus Nerve Reduces Tumor Necrosis Factor in Endotoxemia
JAK/STAT1 signaling promotes HMGB1 hyperacetylation and nuclear translocation
α7 Nicotinic Acetylcholine Receptor Signaling Inhibits Inflammasome Activation by Preventing Mitochondrial DNA Release
Expression of Concern: <scp>HMGB</scp>1 mediates splenomegaly and expansion of splenic <scp>CD</scp>11b+ <scp>L</scp>y‐6<scp>C</scp><sup>high</sup> inflammatory monocytes in murine sepsis survivors
Novel role of PKR in inflammasome activation and HMGB1 release
Renal expression and serum levels of high mobility group box 1 protein in lupus nephritis