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William A. Freed-Pastor

Broad Institute ·
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Area of research
Oncology · Immunology
Research interest
Research interests include Pancreatic and Hepatic Oncology Research, Cancer Immunotherapy and Biomarkers, Immunotherapy and Immune Responses, and Cancer Genomics and Diagnostics.
h-index
20
citations
4,848
works
58
NIH funding
primary concept
Biology
email

Recent publications

Pancreatic cancer–restricted cryptic antigens are targets for T cell recognition
Science 2025cited by 54position: lastdoi
A prime editor mouse to model a broad spectrum of somatic mutations in vivo
Nature Biotechnology 2023cited by 57position: middledoi
Single-nucleus and spatial transcriptome profiling of pancreatic cancer identifies multicellular dynamics associated with neoadjuvant treatment
Nature Genetics 2022cited by 397position: middledoi
Lineage tracing reveals the phylodynamics, plasticity, and paths of tumor evolution
Cell 2022cited by 331position: middledoi
Deciphering the immunopeptidome in vivo reveals new tumour antigens
Nature 2022cited by 103position: middledoi
Conventional type I dendritic cells maintain a reservoir of proliferative tumor-antigen specific TCF-1+ CD8+ T cells in tumor-draining lymph nodes
Immunity 2021cited by 269position: middledoi
The CD155/TIGIT axis promotes and maintains immune evasion in neoantigen-expressing pancreatic cancer
Cancer Cell 2021cited by 259position: firstdoi
Measuring kinetics and metastatic propensity of CTCs by blood exchange between mice
Nature Communications 2021cited by 40position: middledoi
Abstract PO-063: Functional interrogation of immune escape in neoantigen-expressing pancreatic cancer identifies a critical role for the CD155/TIGIT axis
Cancer Research 2021cited by 0position: firstdoi
p53 Represses the Mevalonate Pathway to Mediate Tumor Suppression
Cell 2018cited by 449position: middledoi
Accurate and sensitive quantification of protein-DNA binding affinity
Proceedings of the National Academy of Sciences 2018cited by 129position: middledoi
Quantitative Analysis of the DNA Methylation Sensitivity of Transcription Factor Complexes
Cell Reports 2017cited by 139position: middledoi
The p53 C Terminus Controls Site-Specific DNA Binding and Promotes Structural Changes within the Central DNA Binding Domain
Molecular Cell 2015cited by 120position: middledoi
Mutant p53 Disrupts Mammary Tissue Architecture via the Mevalonate Pathway
Cell 2012cited by 860position: firstdoi

Grants

No grants ingested yet.

Frequent collaborators

Tyler Jacks · Howard Hughes Medical Institute6 papers (2021–2022)Carol Prives · Columbia University4 papers (2012–2018)William M. Rideout · Massachusetts Institute of Technology3 papers (2021–2022)Peter M.K. Westcott · Cold Spring Harbor Laboratory3 papers (2021–2022)George Eng · Massachusetts Institute of Technology3 papers (2021–2021)Alex M. Jaeger · Moffitt Cancer Center3 papers (2021–2022)William L. Hwang · Harvard University3 papers (2021–2021)Oleg Laptenko · Columbia University3 papers (2015–2018)Jason M. Schenkel · The University of Texas MD Anderson Cancer Center3 papers (2021–2022)Sean-Luc Shanahan · Massachusetts Institute of Technology2 papers (2021–2022)Zackery A. Ely · Broad Institute2 papers (2021–2021)Santiago Naranjo · The University of Texas Health Science Center2 papers (2022–2022)Arjun Bhutkar · Massachusetts Institute of Technology2 papers (2021–2021)Aviv Regev · Moscow Institute of Thermal Technology2 papers (2021–2021)Kim L. Mercer · Massachusetts Institute of Technology2 papers (2021–2021)Megan L. Burger · Geisinger Wyoming Valley Medical Center2 papers (2021–2021)Richard S. Mann · Columbia University Irving Medical Center2 papers (2017–2018)Laurens J. Lambert · Massachusetts Institute of Technology2 papers (2021–2021)Anthony M. Barsotti · Columbia University2 papers (2012–2018)Nimisha B. Pattada · University of Pennsylvania2 papers (2021–2021)
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