Barbara Keßler

Reduced Muscle Force in Dystrophic <i>DMD</i> Δ52 Pigs Is Incompletely Restored by Systemic Transcript Reframing ( <i>DMD</i> Δ51–52)

What Genetic Modifications of Source Pigs Are Essential and Sufficient for Cell, Tissue, and Organ Xenotransplantation?

Systemic deletion of <i>DMD</i> exon 51 rescues clinically severe Duchenne muscular dystrophy in a pig model lacking <i>DMD</i> exon 52.

Early disruption of photoreceptor cell architecture and loss of vision in a humanized pig model of usher syndromes

Early disruption of photoreceptor cell architecture and loss of vision in a humanized pig model of usher syndromes.

Growth hormone receptor knockout to reduce the size of donor pigs for preclinical xenotransplantation studies.

Sequential in vivo labeling of insulin secretory granule pools in <i>INS</i>-<i>SNAP</i> transgenic pigs.

A decade of experience with genetically tailored pig models for diabetes and metabolic research

A decade of experience with genetically tailored pig models for diabetes and metabolic research.

Viable pigs after simultaneous inactivation of porcine MHC class I and three xenoreactive antigen genes GGTA1, CMAH and B4GALNT2

Consistent success in life-supporting porcine cardiac xenotransplantation

Efficient production of multi-modified pigs for xenotransplantation by ‘combineering’, gene stacking and gene editing

Ubiquitous LEA29Y Expression Blocks T Cell Co-Stimulation but Permits Sexual Reproduction in Genetically Modified Pigs

361 GROWTH HORMONE RECEPTOR MUTANT PIGS PRODUCED BY USING THE CLUSTERED REGULARLY INTERSPACED SHORT PALINDROMIC REPEATS (CRISPR) AND CRISPR-ASSOCIATED SYSTEMS IN IN VITRO-PRODUCED ZYGOTES

Regulatory Sequences of the Porcine THBD Gene Facilitate Endothelial-Specific Expression of Bioactive Human Thrombomodulin in Single- and Multitransgenic Pigs

322 POTENTIAL OF PRIMARY PORCINE KIDNEY CELLS FOR THE GENERATION OF TRANSGENIC PIGS VIA SOMATIC CELL NUCLEAR TRANSFER