Area of research
Endocrine and Autonomic Systems · Physiology
Research interest
Research interests include Regulation of Appetite and Obesity, Adipose Tissue and Metabolism, Birth, Development, and Health, and Connective tissue disorders research.
ATAD3A has a scaffolding role regulating mitochondria inner membrane structure and protein assembly
Cardiomyopathic mutations in essential light chain reveal mechanisms regulating the super relaxed state of myosin
Enhanced glycolysis and GSK3 inactivation promote brain metabolic adaptations following neuronal mitochondrial stress
Respiratory supercomplexes act as a platform for complex III‐mediated maturation of human mitochondrial complexes I and IV
Impaired mitophagy links mitochondrial disease to epithelial stress in methylmalonyl-CoA mutase deficiency
Myopathy reversion in mice after restauration of mitochondrial complex I
Simultaneous nicotine and oral contraceptive exposure alters brain energy metabolism and exacerbates ischemic stroke injury in female rats
Metformin delays neurological symptom onset in a mouse model of neuronal complex I deficiency
Mitochondrial disease disrupts hepatic allostasis and lowers the threshold for immune-mediated liver toxicity
Nicotine Exposure Along with Oral Contraceptive Treatment in Female Rats Exacerbates Post-cerebral Ischemic Hypoperfusion Potentially via Altered Histamine Metabolism
Hypoxia Promotes Mitochondrial Complex I Abundance via HIF-1α in Complex III and Complex IV Deficient Cells
Photobiomodulation enhancement of cell proliferation at 660 nm does not require cytochrome c oxidase
ATAD3 controls mitochondrial cristae structure in mouse muscle, influencing mtDNA replication and cholesterol levels
Overexpression of <scp>PGC</scp>‐1α in aging muscle enhances a subset of young‐like molecular patterns
The Organization of Mitochondrial Supercomplexes is Modulated by Oxidative Stress In Vivo in Mouse Models of Mitochondrial Encephalopathy
Cytochrome c Oxidase Activity Is a Metabolic Checkpoint that Regulates Cell Fate Decisions During T Cell Activation and Differentiation
Respiration-Deficient Astrocytes Survive As Glycolytic Cells<i>In Vivo</i>
The CoQH2/CoQ Ratio Serves as a Sensor of Respiratory Chain Efficiency
Enhanced Transcriptional Activity and Mitochondrial Localization of STAT3 Co-induce Axon Regrowth in the Adult Central Nervous System
Pioglitazone ameliorates the phenotype of a novel Parkinson’s disease mouse model by reducing neuroinflammation
Sustained AMPK activation improves muscle function in a mitochondrial myopathy mouse model by promoting muscle fiber regeneration
GSNOR Deficiency Enhances <i>In Situ</i> Skeletal Muscle Strength, Fatigue Resistance, and RyR1 S-Nitrosylation Without Impacting Mitochondrial Content and Activity
Cytochrome c oxidase deficiency accelerates mitochondrial apoptosis by activating ceramide synthase 6
The Mitochondrial Metallochaperone SCO1 Is Required to Sustain Expression of the High-Affinity Copper Transporter CTR1 and Preserve Copper Homeostasis
Mitochondrial Diseases Part I: Mouse models of OXPHOS deficiencies caused by defects in respiratory complex subunits or assembly factors
Mitochondrial Diseases Part II: Mouse models of OXPHOS deficiencies caused by defects in regulatory factors and other components required for mitochondrial function
Blockage of the Neonatal Leptin Surge Affects the Gene Expression of Growth Factors, Glial Proteins, and Neuropeptides Involved in the Control of Metabolism and Reproduction in Peripubertal Male and Female Rats
Mitochondrial Diseases Part III: Therapeutic interventions in mouse models of OXPHOS deficiencies
Periodic Estrogen Receptor-Beta Activation: A Novel Approach to Prevent Ischemic Brain Damage
Partial complex I deficiency due to the CNS conditional ablation of Ndufa5 results in a mild chronic encephalopathy but no increase in oxidative damage