Area of research
Pulmonary and Respiratory Medicine · Cancer Research
Research interest
Research focused on Pulmonary hypertension and Terahertz radiation, with related work in Gene knockdown, Hypoxia (environmental), microRNA. Notable publications include 'MicroRNAs in Pulmonary Arterial Hypertension', 'Endothelial and Smooth Muscle Cell Interactions in the Pathobiology of Pulmonary Hypertension', and 'The Sphingosine Kinase 1/Sphingosine-1-Phosphate Pathway in Pulmonary Arterial Hypertension'.
Current challenges and future directions for engineering extracellular vesicles for heart, lung, blood and sleep diseases
MicroRNA-212-5p, an anti-proliferative miRNA, attenuates hypoxia and sugen/hypoxia-induced pulmonary hypertension in rodents
Extracellular vesicles derived from endothelial cells in hypoxia contribute to pulmonary artery smooth muscle cell proliferation in‐vitro and pulmonary hypertension in mice
Smooth muscle cell-specific FoxM1 controls hypoxia-induced pulmonary hypertension
PAI-1 is a novel component of the miR-17~92 signaling that regulates pulmonary artery smooth muscle cell phenotypes
Downregulation of PKCζ/Pard3/Pard6b is responsible for lung adenocarcinoma cell EMT and invasion
Endothelial and Smooth Muscle Cell Interactions in the Pathobiology of Pulmonary Hypertension
miR‐17/20 Controls Prolyl Hydroxylase 2 (PHD2)/Hypoxia‐Inducible Factor 1 (HIF1) to Regulate Pulmonary Artery Smooth Muscle Cell Proliferation
Loss of MicroRNA-17∼92 in Smooth Muscle Cells Attenuates Experimental Pulmonary Hypertension via Induction of PDZ and LIM Domain 5
Active terahertz device based on optically controlled organometal halide perovskite
High-efficiency THz modulator based on phthalocyanine-compound organic films
Optically-controlled metamaterial absorber based on hybrid structure
MicroRNAs in Pulmonary Arterial Hypertension
The Sphingosine Kinase 1/Sphingosine-1-Phosphate Pathway in Pulmonary Arterial Hypertension
Conjugated polymer-based broadband terahertz wave modulator
Intratracheal Instillation of High Dose Adenoviral Vectors Is Sufficient to Induce Lung Injury and Fibrosis in Mice
Adenosine Monophosphate–Activated Protein Kinase Is Required for Pulmonary Artery Smooth Muscle Cell Survival and the Development of Hypoxic Pulmonary Hypertension
cAMP-dependent protein kinase is essential for hypoxia-mediated epithelial–mesenchymal transition, migration, and invasion in lung cancer cells
Knockdown of von Hippel–Lindau protein decreases lung cancer cell proliferation and colonization