Area of research
Cancer Research · Surgery
Research interest
Research interests include MicroRNA in disease regulation, Cervical and Thoracic Myelopathy, Spine and Intervertebral Disc Pathology, and Cancer-related molecular mechanisms research.
Immuno-engineered macrophage membrane-coated nanodrug to restore immune balance for rheumatoid arthritis treatment
Parathyroid hormone treatment partially reverses endplate remodeling and attenuates low back pain in animal models of spine degeneration
Exosomes, a New Star for Targeted Delivery
m<sup>6</sup>A modification of lncRNA <i>PCAT6</i> promotes bone metastasis in prostate cancer through <i>IGF2BP2</i>‐mediated <i>IGF1R</i> mRNA stabilization
MYBL2 disrupts the Hippo-YAP pathway and confers castration resistance and metastatic potential in prostate cancer
Hyaluronic acid-based interpenetrating network hydrogel as a cell carrier for nucleus pulposus repair
Spinal Cord Parenchyma Vascular Redistribution Underlies Hemodynamic and Neurophysiological Changes at Dynamic Neck Positions in Cervical Spondylotic Myelopathy
SMAD3/SP1 complex‐mediated constitutive active loop between lncRNA PCAT7 and TGF‐β signaling promotes prostate cancer bone metastasis
Ectopic Expression of miR-532-3p Suppresses Bone Metastasis of Prostate Cancer Cells via Inactivating NF-κB Signaling
miR-582-3p and miR-582-5p Suppress Prostate Cancer Metastasis to Bone by Repressing TGF-β Signaling
miR-204-5p Represses Bone Metastasis via Inactivating NF-κB Signaling in Prostate Cancer
Copy number gain of ZEB1 mediates a double-negative feedback loop with miR-33a-5p that regulates EMT and bone metastasis of prostate cancer dependent on TGF-β signaling
A New Diagnostic Medium for Cervical Spondylotic Myelopathy: Dynamic Somatosensory Evoked Potentials
Wnt5a induces and maintains prostate cancer cells dormancy in bone
Downregulation of miR-133a-3p promotes prostate cancer bone metastasis via activating PI3K/AKT signaling
Oncogenic miR-210-3p promotes prostate cancer cell EMT and bone metastasis via NF-κB signaling pathway
Downregulation of miR-141-3p promotes bone metastasis via activating NF-κB signaling in prostate cancer
Double-negative feedback loop between ZEB2 and miR-145 regulates epithelial-mesenchymal transition and stem cell properties in prostate cancer cells
Loss of miR-100 enhances migration, invasion, epithelialmesenchymal transition and stemness properties in prostate cancer cells through targeting Argonaute 2
Wild-type p53 suppresses the epithelial-mesenchymal transition and stemness in PC-3 prostate cancer cells by modulating miR-145
HEF1 promotes epithelial mesenchymal transition and bone invasion in prostate cancer under the regulation of microRNA‐145
miR-143 and miR-145 inhibit stem cell characteristics of PC-3 prostate cancer cells