Area of research
Genetics · Immunology
Research interest
Research focused on Immunology and Leukemia, with related work in Neutropenia, Immunophenotyping, CD8. Notable publications include 'Intrinsic and extrinsic mechanisms contribute to maintain the JAK/STAT pathway aberrantly activated in T-type large granular lymphocyte leukemia', 'STAT3 mutation impacts biological and clinical features of T-LGL leukemia', and 'Stat3 mutations impact on overall survival in large granular lymphocyte leukemia: a single-center experience of 205 patients'.
Not all LGL leukemias are created equal
Single-cell characterization of leukemic and non-leukemic immune repertoires in CD8+ T-cell large granular lymphocytic leukemia
Tγδ LGLL identifies a subset with more symptomatic disease: analysis of an international cohort of 137 patients
Hypocellular myelodysplastic syndromes (h-MDS): from clinical description to immunological characterization in the Italian multi-center experience
All that glitters is not LGL Leukemia
Neutropenia and Large Granular Lymphocyte Leukemia: From Pathogenesis to Therapeutic Options
Insights Into Genetic Landscape of Large Granular Lymphocyte Leukemia
A high definition picture of somatic mutations in chronic lymphoproliferative disorder of natural killer cells
Identification of the true hyperdiploid multiple myeloma subset by combining conventional karyotyping and FISH analysis
Stat3 mutations impact on overall survival in large granular lymphocyte leukemia: a single-center experience of 205 patients
Identification of a <i>miR-146b</i>-Fas ligand axis in the development of neutropenia in T large granular lymphocyte leukemia
Dominant cytotoxic NK cell subset within CLPD-NK patients identifies a more aggressive NK cell proliferation
<i>STAT3</i> mutation impacts biological and clinical features of T-LGL leukemia
A Pyrazolo[3,4-<i>d</i>]pyrimidine compound inhibits Fyn phosphorylation and induces apoptosis in natural killer cell leukemia
TL1A/DR3 axis involvement in the inflammatory cytokine network during pulmonary sarcoidosis
Intrinsic and extrinsic mechanisms contribute to maintain the JAK/STAT pathway aberrantly activated in T-type large granular lymphocyte leukemia
HS1, a Lyn Kinase Substrate, Is Abnormally Expressed in B-Chronic Lymphocytic Leukemia and Correlates with Response to Fludarabine-Based Regimen