Area of research
Endocrinology, Diabetes and Metabolism · Oncology
Research interest
Research interests include Thyroid Cancer Diagnosis and Treatment, Cancer-related Molecular Pathways, Thyroid Disorders and Treatments, and Vitamin D Research Studies.
SGK3 promoter deletion in late-onset hypophosphatemic rickets, a possible genetic cause of the disease.
Molecular Genetics of 1α-Hydroxylase Deficiency in the Saudi Population.
Genome-wide transcriptome analysis and drug target discovery reveal key genes and pathways in thyroid cancer metastasis.
Identification of novel prognostic biomarkers for thyroid cancer by integrated transcriptome analysis of metastasis-associated genes.
Vonoprazan‐based therapies versus PPI‐based therapies in patients with <i>H. pylori</i> infection: Systematic review and meta‐analyses of randomized controlled trials
Tune-A-Video: One-Shot Tuning of Image Diffusion Models for Text-to-Video Generation
Molecular Genetics of Diffuse Sclerosing Papillary Thyroid Cancer.
β-catenin attenuation leads to up-regulation of activating NKG2D ligands and tumor regression in <i>Braf<sup>V600E</sup></i>-driven thyroid cancer cells.
Fig. S1 from β-Catenin Attenuation Inhibits Tumor Growth and Promotes Differentiation in a BRAF<sup>V600E</sup>-Driven Thyroid Cancer Animal Model
Fig. S1 from β-Catenin Attenuation Inhibits Tumor Growth and Promotes Differentiation in a BRAF<sup>V600E</sup>-Driven Thyroid Cancer Animal Model
Fig. S2 from β-Catenin Attenuation Inhibits Tumor Growth and Promotes Differentiation in a BRAF<sup>V600E</sup>-Driven Thyroid Cancer Animal Model
Fig. S2 from β-Catenin Attenuation Inhibits Tumor Growth and Promotes Differentiation in a BRAF<sup>V600E</sup>-Driven Thyroid Cancer Animal Model
Data from β-Catenin Attenuation Inhibits Tumor Growth and Promotes Differentiation in a BRAF<sup>V600E</sup>-Driven Thyroid Cancer Animal Model
Data from β-Catenin Attenuation Inhibits Tumor Growth and Promotes Differentiation in a BRAF<sup>V600E</sup>-Driven Thyroid Cancer Animal Model
A Spatiotemporal Graph Attention Network Based on Synchronization for Epileptic Seizure Prediction
Insights of Noncanonical Splice-site Variants on RNA Splicing in Patients With Congenital Hypothyroidism.
A novel claudin-10 mutation with a unique mechanism in two unrelated families with HELIX syndrome.
Mutation spectrum of EXT1 and EXT2 in the Saudi patients with hereditary multiple exostoses.
β-Catenin Attenuation Inhibits Tumor Growth and Promotes Differentiation in a BRAF<sup>V600E</sup>-Driven Thyroid Cancer Animal Model.
Mutation of SGK3, a Novel Regulator of Renal Phosphate Transport, Causes Autosomal Dominant Hypophosphatemic Rickets.
Molecular Analysis of <i>CYP27B1</i> Mutations in Vitamin D-Dependent Rickets Type 1A: c.590G > A (p.G197D) Missense Mutation Causes a RNA Splicing Error.
NOVEL <i>VDR</i> MUTATIONS IN PATIENTS WITH VITAMIN D-DEPENDENT RICKETS TYPE 2A: A MILD DISEASE PHENOTYPE CAUSED BY A NOVEL CANONICAL SPLICE-SITE MUTATION.
Functional analysis of 22 splice-site mutations in the PHEX, the causative gene in X-linked dominant hypophosphatemic rickets.
Cantharidin inhibits cell proliferation and induces apoptosis through G2/M phase cell cycle arrest in hepatocellular carcinoma stem cells
Computational identification of specific splicing regulatory elements from RNA-seq in lung cancer.
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