Area of research
Speech and Hearing · Health, Toxicology and Mutagenesis
Research interest
Research focused on Oxidative stress and Environmental health, with related work in Inflammation, Disease, Liraglutide. Notable publications include 'Vascular Inflammation and Oxidative Stress: Major Triggers for Cardiovascular Disease', 'Endothelial GLP-1 (Glucagon-Like Peptide-1) Receptor Mediates Cardiovascular Protection by Liraglutide In Mice With Experimental Arterial Hypertension', and 'Crucial role for Nox2 and sleep deprivation in aircraft noise-induced vascular and cerebral oxidative stress, inflammation, and gene regulation'.
Short-term aircraft noise stress induces a fundamental metabolic shift in heart proteome and metabolome that bears the hallmarks of cardiovascular disease
The dynamics of hematopoiesis over the human lifespan
Exposure to aircraft noise exacerbates cardiovascular and oxidative damage in three mouse models of diabetes
Maturation and persistence of CAR T cells derived from human pluripotent stem cells via chemical inhibition of G9a/GLP
The epigenetic state of the cell of origin defines mechanisms of leukemogenesis
Health position paper and redox perspectives - Disease burden by transportation noise
Noise and Air Pollution as Risk Factors for Hypertension: Part I—Epidemiology
Mitigation of aircraft noise-induced vascular dysfunction and oxidative stress by exercise, fasting, and pharmacological α1AMPK activation: molecular proof of a protective key role of endothelial α1AMPK against environmental noise exposure
Effects of aircraft noise cessation on blood pressure, cardio- and cerebrovascular endothelial function, oxidative stress, and inflammation in an experimental animal model
RUNX1 mutations mitigate quiescence to promote transformation of hematopoietic progenitors in Fanconi anemia
Cerebral consequences of environmental noise exposure
Protective actions of nuclear factor erythroid 2-related factor 2 (NRF2) and downstream pathways against environmental stressors
Redox Regulatory Changes of Circadian Rhythm by the Environmental Risk Factors Traffic Noise and Air Pollution
Long-Term Effects of Aircraft Noise Exposure on Vascular Oxidative Stress, Endothelial Function and Blood Pressure: No Evidence for Adaptation or Tolerance Development
Accelerated Aging and Age-Related Diseases (CVD and Neurological) Due to Air Pollution and Traffic Noise Exposure
GLP-1 Analog Liraglutide Improves Vascular Function in Polymicrobial Sepsis by Reduction of Oxidative Stress and Inflammation
Thiol-based redox-active proteins as cardioprotective therapeutic agents in cardiovascular diseases
Aircraft noise exposure drives the activation of white blood cells and induces microvascular dysfunction in mice
Physical Activity in Polluted Air—Net Benefit or Harm to Cardiovascular Health? A Comprehensive Review
Ablation of lysozyme M-positive cells prevents aircraft noise-induced vascular damage without improving cerebral side effects
Noise-Induced Vascular Dysfunction, Oxidative Stress, and Inflammation Are Improved by Pharmacological Modulation of the NRF2/HO-1 Axis
Redox Switches in Noise-Induced Cardiovascular and Neuronal Dysregulation
In vivo analysis of noise dependent activation of white blood cells and microvascular dysfunction in mice
The AMP-Activated Protein Kinase Plays a Role in Antioxidant Defense and Regulation of Vascular Inflammation
Exacerbation of adverse cardiovascular effects of aircraft noise in an animal model of arterial hypertension
Environmental Factors Such as Noise and Air Pollution and Vascular Disease
Environmental aircraft noise aggravates oxidative DNA damage, granulocyte oxidative burst and nitrate resistance in<i>Ogg1</i><sup>–/–</sup>mice
Vascular Inflammation and Oxidative Stress: Major Triggers for Cardiovascular Disease
Endothelial GLP-1 (Glucagon-Like Peptide-1) Receptor Mediates Cardiovascular Protection by Liraglutide In Mice With Experimental Arterial Hypertension
Short-term e-cigarette vapour exposure causes vascular oxidative stress and dysfunction: evidence for a close connection to brain damage and a key role of the phagocytic NADPH oxidase (NOX-2)