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Julie E. Buring

Collaborative Research Group · AU
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Area of research
Epidemiology · Genetics
Research interest
has been on the epidemiology of chronic diseases, particularly cardiovascular disease and cancer, and especially among women. I am Principal Investigator or co-Principal Investigator of four large-scale randomized clinical trials or their observational follow-up: the Women's Health Study, a primary prevention trial that evaluated the balance of benefits and risks of low dose aspirin and vitamin E on cardiovascular disease and cancer among 40,000 female health professionals; the Women's Antioxidant Cardiovascular Study, a secondary prevention trial that evaluated the roles of vitamin E, vitamin C, beta-carotene and folic acid/B6/B12 among 8,000 women with a prior history of heart disease; the Physicians' Health Study II, that evaluated vitamin E, vitamin C, beta-carotene and a multivitamin
h-index
179
citations
129,772
works
1,308
NIH funding
primary concept
email

Recent publications

Hearing Intervention, Social Isolation, and Loneliness
JAMA Internal Medicine 2025cited by 10position: middledoi
Different diabetes types and pancreatic ductal adenocarcinoma: a Mendelian randomization and pathway/gene-set analysis
JNCI: Journal of the National Cancer Institute 2025cited by 1position: contributordoi
Pre-diagnostic circulating bile acid concentrations and liver cancer risk: a nested case-control analysis of 12 cohorts
JNCI Cancer Spectrum 2025cited by 1position: contributordoi
Unsupervised Learning Analysis of Triglycerides, Inflammation, Cholesterol, and the Risks of Incident Cardiovascular Disease and Type 2 Diabetes in the Women's Health Study
Journal of the American Heart Association 2025cited by 1position: contributordoi
Different diabetes types and pancreatic ductal adenocarcinoma: a Mendelian randomization and pathway/gene-set analysis
JNCI Journal of the National Cancer Institute 2025cited by 1position: middledoi
Supplementary Table S3 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
2025cited by 0position: contributordoi
Supplementary Table S5 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Supplementary Table S4 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Supplementary Table S10 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Supplementary Figure S1 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
2025cited by 0position: contributordoi
Supplementary Table S7 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Supplementary Figure S2 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Supplementary Table S4 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
2025cited by 0position: contributordoi
Supplementary Table S2 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
2025cited by 0position: contributordoi
Supplementary Figure S1 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Supplementary Table S9 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Supplementary Table S5 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
2025cited by 0position: contributordoi
Supplementary Table S6 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Supplementary Figure S2 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
2025cited by 0position: contributordoi
Supplementary Table S8 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Supplementary Table S1 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
2025cited by 0position: contributordoi
Supplementary Table S3 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Supplementary Table S2 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Supplementary Table S1 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
2025cited by 0position: contributordoi
Understanding the genetic complexity of puberty timing across the allele frequency spectrum
Nature Genetics 2024cited by 44position: middledoi
Self-reported oral health is associated with systemic health outcomes and all-cause mortality
The Journal of the American Dental Association 2024cited by 21position: contributordoi
Intake of Blueberries, Anthocyanins, and Risk of Eye Disease in Women
Journal of Nutrition 2024cited by 12position: middledoi
Associations of Vitamin D-Related Biomarkers With Hypertension and the Renin–Angiotensin System in Men and Women
American Journal of Hypertension 2024cited by 3position: contributordoi
Associations of Everyday Discrimination With Insomnia and Short Sleep Duration Among Older Women.
2024cited by 2position: contributordoi
Genome-Wide Analysis to Assess if Heavy Alcohol Consumption Modifies the Association between SNPs and Pancreatic Cancer Risk
Cancer Epidemiology, Biomarkers & Prevention 2024cited by 1position: contributordoi

Grants

No grants ingested yet.

Frequent collaborators

JoAnn E. Manson · The University of Texas Health Science Center at Houston26 papers (2012–2024) · 22 papers (2020–2025)I‐Min Lee · Harvard University17 papers (2013–2024)Howard D Sesso · Mount Sinai Hospital11 papers (2020–2024)I-Min Lee · University of Washington10 papers (2020–2024) · 10 papers (2020–2024)Nancy R. Cook · Harvard University10 papers (2013–2023)Charles S. Fuchs · Kaiser Permanente South San Francisco Medical Center8 papers (2013–2024)Peter Kraft · National Cancer Institute8 papers (2013–2024)Paul M. Ridker · Gentofte Hospital7 papers (2012–2025) · 7 papers (2023–2024)Aladdin H. Shadyab · University of San Diego7 papers (2020–2024)Jean Wactawski-Wende · New York College of Health Professions7 papers (2023–2024)Meir J. Stampfer · Harvard University7 papers (2013–2024)Jihye Kim · W. L. Gore & Associates (United States)6 papers (2023–2024)Fred K. Tabung · The Ohio State University6 papers (2023–2024) · 6 papers (2023–2024)Brian M. Wolpin · Dana-Farber Brigham Cancer Center6 papers (2023–2024)Chen Yuan · Harvard University6 papers (2023–2024)Nazmus Saquib · University of Washington6 papers (2023–2024)
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