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Mathew Wallis

Wenzhou Medical University · CN
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Area of research
Genetics · Molecular Biology
Research interest
Research focused on Genetics and Breast cancer, with related work in Penetrance, Trinucleotide repeat expansion, Disease. Notable publications include 'An intronic GAA repeat expansion in FGF14 causes the autosomal-dominant adult-onset ataxia SCA27B/ATX-FGF14', 'Integrated multi-omics for rapid rare disease diagnosis on a national scale', and 'Heritable DNA methylation marks associated with susceptibility to breast cancer'.
h-index
citations
704
works
10
NIH funding
primary concept
email

Recent publications

Untargeted proteomics enables ultra-rapid variant prioritisation in mitochondrial and other rare diseases
Genome Medicine 2025cited by 11position: middledoi
Cancer Risks Associated With <i>TP53</i> Pathogenic Variants: Maximum Likelihood Analysis of Extended Pedigrees for Diagnosis of First Cancers Beyond the Li-Fraumeni Syndrome Spectrum
JCO Precision Oncology 2024cited by 23position: middledoi
Implementation and Evaluation of a National Multidisciplinary Kidney Genetics Clinic Network Over 10 Years
Kidney International Reports 2024cited by 22position: middledoi
Genomic Testing in Patients with Kidney Failure of an Unknown Cause
Clinical Journal of the American Society of Nephrology 2024cited by 19position: middledoi
Integrated multi-omics for rapid rare disease diagnosis on a national scale
Nature Medicine 2023cited by 133position: middledoi
An intronic GAA repeat expansion in FGF14 causes the autosomal-dominant adult-onset ataxia SCA27B/ATX-FGF14
The American Journal of Human Genetics 2022cited by 163position: middledoi
The Australian Reproductive Genetic Carrier Screening Project (Mackenzie’s Mission): Design and Implementation
Journal of Personalized Medicine 2022cited by 60position: middledoi
Germline variants in tumor suppressor FBXW7 lead to impaired ubiquitination and a neurodevelopmental syndrome
The American Journal of Human Genetics 2022cited by 41position: middledoi
Heritable DNA methylation marks associated with susceptibility to breast cancer
Nature Communications 2018cited by 116position: middledoi
CCDC22 deficiency in humans blunts activation of proinflammatory NF-κB signaling
Journal of Clinical Investigation 2013cited by 116position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

· 1 papers (2025–2025)Haiying Li · Second Xiangya Hospital of Central South University1 papers (2013–2013)Anand Vasudevan · Royal Women's Hospital1 papers (2025–2025) · 1 papers (2025–2025)Xicheng Mao · University of Alabama at Birmingham1 papers (2013–2013) · 1 papers (2025–2025)Marco Y. Hein · Vienna Biocenter1 papers (2013–2013)Iram Waris Zaidi · The University of Texas Southwestern Medical Center1 papers (2013–2013)Natalie B. Tan · Victorian Clinical Genetics Services1 papers (2025–2025)Luke E. Formosa · Monash University1 papers (2025–2025)Hans‐Hilger Ropers · Johannes Gutenberg University Mainz1 papers (2013–2013)Mary‐Louise Freckmann · UNSW Sydney1 papers (2025–2025)Jozef Gécz · SA Health1 papers (2013–2013) · 1 papers (2013–2013) · 1 papers (2013–2013)Petro Starokadomskyy · Southwestern Medical Center1 papers (2013–2013) · 1 papers (2025–2025)Bryony Ryder · Hospital for Sick Children1 papers (2025–2025) · 1 papers (2025–2025) · 1 papers (2025–2025)
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