Area of research
Cellular and Molecular Neuroscience · Physiology
Research interest
Research interests include Pain Mechanisms and Treatments, Receptor Mechanisms and Signaling, Neuropeptides and Animal Physiology, and Neurotransmitter Receptor Influence on Behavior.
A µ-opioid receptor superagonist analgesic with minimal adverse effects
HDAC6 inhibition ameliorates sensory hypersensitivity and reduces immune cell signatures in the dorsal root ganglia in murine chronic pain models
Annulus Fibrosus Injury Induces Acute Neuroinflammation and Chronic Glial Response in Dorsal Root Ganglion and Spinal Cord—An In Vivo Rat Discogenic Pain Model
tRNA epitranscriptomic alterations associated with opioid-induced reward-seeking and long-term opioid withdrawal in male mice
Nucleus accumbens myocyte enhancer factor 2C mediates the maintenance of peripheral nerve injury–induced physiological and behavioral maladaptations
RGS4 Actions in Mouse Prefrontal Cortex Modulate Behavioral and Transcriptomic Responses to Chronic Stress and Ketamine
Author Correction: Oxycodone withdrawal induces HDAC1/HDAC2-dependent transcriptional maladaptations in the reward pathway in a mouse model of peripheral nerve injury
SARS-CoV-2 airway infection results in the development of somatosensory abnormalities in a hamster model
Oxycodone withdrawal induces HDAC1/HDAC2-dependent transcriptional maladaptations in the reward pathway in a mouse model of peripheral nerve injury
Spinal Cord Sensitization and Spinal Inflammation from an In Vivo Rat Endplate Injury Associated with Painful Intervertebral Disc Degeneration
Tianeptine promotes lasting antiallodynic effects in a mouse model of neuropathic pain
SARS-CoV-2 and Influenza A Virus Induce Longitudinal Transcriptomic Changes in Hamster Spinal Cord Tissue
tRNA epitranscriptomic alterations associated with opioid-induced reward-seeking and long-term opioid withdrawal
SARS-CoV-2 infection in hamsters and humans results in lasting and unique systemic perturbations after recovery
Comparative Transcriptional Analyses in the Nucleus Accumbens Identifies RGS2 as a Key Mediator of Depression-Related Behavior
A Regional and Projection-Specific Role of RGSz1 in the Ventrolateral Periaqueductal Grey in the Modulation of Morphine Reward
SARS-CoV-2 Airway Infection Results in Time-dependent Sensory Abnormalities in a Hamster Model
Chronic pain‐mediated Regulator of G protein signaling 4 (RGS4) gene expression in superficial dorsal horn of spinal cord
Microglia and macrophages promote corralling, wound compaction and recovery after spinal cord injury via Plexin-B2
HDAC6-selective inhibitors decrease nerve-injury and inflammation-associated mechanical hypersensitivity in mice
Regulators of G Protein Signaling in Analgesia and Addiction
Reasons for Failed Trials of Disease-Modifying Treatments for Alzheimer Disease and Their Contribution in Recent Research
The Mesolimbic Dopamine System in Chronic Pain and Associated Affective Comorbidities
Reasons for Failed trials of Disease-modifying Treatments for Alzheimer Disease and their Contribution in Recent Research
RGS4 Maintains Chronic Pain Symptoms in Rodent Models
RGS9‐2 rescues dopamine D2 receptor levels and signaling in DYT1 dystonia mouse models
Suppression of RGSz1 function optimizes the actions of opioid analgesics by mechanisms that involve the Wnt/β-catenin pathway
Neuropathic pain promotes adaptive changes in gene expression in brain networks involved in stress and depression
Brain-Derived Neurotrophic Factor in the Mesolimbic Reward Circuitry Mediates Nociception in Chronic Neuropathic Pain
RGS9-2 Modulates Responses to Oxycodone in Pain-Free and Chronic Pain States