Area of research
Neurology · Complementary and alternative medicine
Research interest
Research interests include Neuroinflammation and Neurodegeneration Mechanisms, Neurological Disease Mechanisms and Treatments, Neurological Disorders and Treatments, and Traditional Chinese Medicine Analysis.
Neuroprotective Effects, Mechanisms of Action and Therapeutic Potential of the Kv7/KCNQ Channel Opener QO-83 in Ischemic Stroke
Dl-3-n-Butylphthalide Promotes Neurogenesis in Ischemic Stroke Mice Through Wnt/β-Catenin Signaling Activation and Neurotrophic Factor Production
Dl‐3‐n‐Butylphthalide Promotes Cortical Angiogenesis via Akt/GSK‐3β Signaling in Ischemic Stroke Mice
Closed-Loop Transcranial Ultrasound Stimulation Based on Deep Learning Effectively Suppresses Epileptic Seizures in Mice
Salidroside promotes angiogenesis after cerebral ischemia in mice through Shh signaling pathway
Anisodine hydrobromide injection promotes neural remodeling and recovery after ischemic stroke in mice
Neuroprotective effects, mechanisms of action and therapeutic potential of the Kv7/KCNQ channel opener QO-83 in ischemic stroke
Dl-3-n-butylphthalide promotes neurogenesis in ischemic stroke mice through Wnt/β- catenin signaling activation and neurotrophic factors production
Cervical alignment and clinical outcome of anterior cervical discectomy and fusion vs. anterior cervical corpectomy and fusion in local kyphotic cervical spondylotic myelopathy
Neuroprotective mechanism of salvianolic acid B against cerebral ischemia–reperfusion injury in mice through downregulation of TLR4, p‐p38MAPK, p‐JNK, NF‐κB, and IL‐1β
Protective effects of low-intensity pulsed ultrasound (LIPUS) against cerebral ischemic stroke in mice by promoting brain vascular remodeling via the inhibition of ROCK1/p-MLC2 signaling pathway
Chrysophanol facilitates long-term neurological recovery through limiting microglia-mediated neuroinflammation after ischemic stroke in mice
Liraglutide Ameliorates Cerebral Ischemia in Mice via Antipyroptotic Pathways
Cerebroprotein Hydrolysate is Involved in Promoting Long-Term Angiogenesis, Vessel Diameter and Density after Cerebral Ischemia in Mice
Neuroprotective effects of zonisamide on cerebral ischemia injury via inhibition of neuronal apoptosis
Atorvastatin alleviates microglia-mediated neuroinflammation via modulating the microbial composition and the intestinal barrier function in ischemic stroke mice
HMGB1 A box protects neurons by potently inhibiting both microglia and T cell-mediated inflammation in a mouse Parkinson’s disease model
A Novel Cerebroprotein Hydrolysate, CH1, Ameliorates Chronic Focal Cerebral Ischemia Injury by Promoting White Matter Integrity via the Shh/Ptch-1/Gli-1 Signaling Pathway
MiR-31 aggravates inflammation and apoptosis in COPD rats via activating the NF-κB signaling pathway
DOAJ (DOAJ: Directory of Open Access Journals) 2020cited by 10position: middle
NG2 glia regulate brain innate immunity via TGF-β2/TGFBR2 axis
Cortical Neuron-Derived Exosomal MicroRNA-181c-3p Inhibits Neuroinflammation by Downregulating CXCL1 in Astrocytes of a Rat Model with Ischemic Brain Injury
Sphingomyelin Synthase 2 Inhibition Ameliorates Cerebral Ischemic Reperfusion Injury Through Reducing the Recruitment of Toll‐Like Receptor 4 to Lipid Rafts
Dl-3-N-Butylphthalide Alleviates the Blood–Brain Barrier Permeability of Focal Cerebral Ischemia Reperfusion in Mice
Tert-butylhydroquinone enhanced angiogenesis and astrocyte activation by activating nuclear factor-E2-related factor 2/heme oxygenase-1 after focal cerebral ischemia in mice
Loss of neuronal CD200 contributed to microglial activation after acute cerebral ischemia in mice
Salvianolic acids enhance cerebral angiogenesis and neurological recovery by activating <scp>JAK</scp>2/<scp>STAT</scp>3 signaling pathway after ischemic stroke in mice
CKII-SIRT1-SM22α loop evokes a self-limited inflammatory response in vascular smooth muscle cells
Salvianolic Acids for Injection (SAFI) promotes functional recovery and neurogenesis via sonic hedgehog pathway after stroke in mice
Accumulation of Smooth Muscle 22α Protein Accelerates Senescence of Vascular Smooth Muscle Cells via Stabilization of p53 In Vitro and In Vivo
EZH2-mediated α-actin methylation needs lncRNA TUG1, and promotes the cortex cytoskeleton formation in VSMCs