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Yunfei Zhang

Shanxi Medical University · CN
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Area of research
Molecular Medicine · Cancer Research
Research interest
Research topics from publications: MBD2 promotes B cell differentiation and BCR signaling in systemic lupus erythematosus by regulating the LEF-1-PTEN-PI3K axis. Representative work: Systemic lupus erythematosus (SLE) is a typical autoimmune disease characterized by the overproduction of autoantibodies and type I interferon, which damages its own tissues, causing multiple organ damage. B cells are thought to play a major role in the pathogenesis of SLE. As a DNA methylation reader, Methyl-CpG-binding domain protein 2 (MBD2) has been extensively studied in the contexts of innate immunity, adaptive immunity, and autoimmune diseases. However, its specific role in B cells and SLE remains unexamined. Herein, we found that MBD2 was highly expressed in B cells of SLE patients and positively correlated with disease activity. Knockout of MBD2 in B cells disturbed B-cell different
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Recent publications

MBD2 promotes B cell differentiation and BCR signaling in systemic lupus erythematosus by regulating the LEF-1-PTEN-PI3K axis
Cell Death and Disease 2025cited by 2position: middledoi

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Frequent collaborators

Haonan Li · Tongji University1 papers (2025–2025)Ying Hu · Shanxi Medical University1 papers (2025–2025)Xiaocui Wang · Anhui Medical University1 papers (2025–2025)Xiansheng Liu · Anhui Medical University1 papers (2025–2025)Shumin Dong · Kunming University of Science and Technology1 papers (2025–2025)Xin Dai · Harbin Medical University1 papers (2025–2025)Yukai Jing · Huazhong University of Science and Technology1 papers (2025–2025)Ze Yan · Shanxi Medical University1 papers (2025–2025)Yufen Wang · University of Southern California1 papers (2025–2025)Bin Wen · Shanxi Medical University1 papers (2025–2025)Cong‐Yi Wang · Wuhan Union Hospital1 papers (2025–2025)Xuemei Duan · Ningbo University1 papers (2025–2025)Yufeng Fan · Shanxi Medical University1 papers (2025–2025)Ruiping Zhang · Jiangsu University1 papers (2025–2025)
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