Area of research
Periodontics · Molecular Biology
Research interest
Research focused on RANKL and Porphyromonas gingivalis, with related work in Dental floss, TLR2, Periodontitis. Notable publications include 'Porphyromonas gingivalis Exacerbates Ligature-Induced, RANKL-Dependent Alveolar Bone Resorption via Differential Regulation of Toll-Like Receptor 2 (TLR2) and TLR4', 'Porphyromonas gingivalis Infection-Associated Periodontal Bone Resorption Is Dependent on Receptor Activator of NF-κB Ligand', and 'B10 Cells Alleviate Periodontal Bone Loss in Experimental Periodontitis'.
Saliva Microbiome Changes in Patients With Periodontitis With and Without Chronic Obstructive Pulmonary Disease
Local promotion of B10 function alleviates experimental periodontitis bone loss through antagonizing RANKL‐expressing neutrophils
Periodontal health: A national cross‐sectional study of knowledge, attitudes and practices for the public oral health strategy in China
Periodontal health: A national cross‐sectional study of knowledge, attitudes and practices for the public oral health strategy in China
2019cited by 2position: middle
B10 Cells Alleviate Periodontal Bone Loss in Experimental Periodontitis
Lipopolysaccharides-Induced Suppression of Innate-Like B Cell Apoptosis Is Enhanced by CpG Oligodeoxynucleotide and Requires Toll-Like Receptors 2 and 4
Porphyromonas gingivalis Exacerbates Ligature-Induced, RANKL-Dependent Alveolar Bone Resorption via Differential Regulation of Toll-Like Receptor 2 (TLR2) and TLR4
Porphyromonas gingivalis Infection-Associated Periodontal Bone Resorption Is Dependent on Receptor Activator of NF-κB Ligand
Activation of Toll‐like receptor 9 inhibits lipopolysaccharide‐induced receptor activator of nuclear factor kappa‐ B ligand expression in rat B lymphocytes