Area of research
Epidemiology · Molecular Biology
Research interest
Research focused on Artemisinin and Autophagy, with related work in TFEB, Mechanism of action, Plasmodium falciparum. Notable publications include 'Haem-activated promiscuous targeting of artemisinin in Plasmodium falciparum', 'Activation of lysosomal function in the course of autophagy via mTORC1 suppression and autophagosome-lysosome fusion', and 'Histone deacetylase inhibitors induce autophagy through FOXO1-dependent pathways'.
Prognostic value of LncRNA PSMA3-AS1 in prostate cancer and its potential regulatory mechanism
The tumor suppressor Parkin exerts anticancer effects through regulating mitochondrial GAPDH activity
The role of remifentanil in regulating mitochondrial autophagy in osteoclasts wasinvestigated based on PINK1/Parkin pathway
ANXA6 suppresses the tumorigenesis of cervical cancer through autophagy induction
Targeting autophagy enhances the anticancer effect of artemisinin and its derivatives
PTEN-L is a novel protein phosphatase for ubiquitin dephosphorylation to inhibit PINK1–Parkin-mediated mitophagy
Importance of TFEB acetylation in control of its transcriptional activity and lysosomal function in response to histone deacetylase inhibitors
Artesunate-induced mitophagy alters cellular redox status
Mechanistic Investigation of the Specific Anticancer Property of Artemisinin and Its Combination with Aminolevulinic Acid for Enhanced Anticolorectal Cancer Activity
Curcumin targets the TFEB-lysosome pathway for induction of autophagy
Mechanism‐Guided Design and Synthesis of a Mitochondria‐Targeting Artemisinin Analogue with Enhanced Anticancer Activity
In situ Proteomic Profiling of Curcumin Targets in HCT116 Colon Cancer Cell Line
Haem-activated promiscuous targeting of artemisinin in Plasmodium falciparum
Histone deacetylase inhibitors induce autophagy through FOXO1-dependent pathways
Mapping sites of aspirin-induced acetylations in live cells by quantitative acid-cleavable activity-based protein profiling (QA-ABPP)
A Quantitative Chemical Proteomics Approach to Profile the Specific Cellular Targets of Andrographolide, a Promising Anticancer Agent That Suppresses Tumor Metastasis
Activation of lysosomal function in the course of autophagy via mTORC1 suppression and autophagosome-lysosome fusion