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Sen Chen

Union Hospital ·
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Area of research
Sensory Systems · Molecular Biology
Research interest
Research focused on Autophagy and Hearing loss, with related work in Cell biology, Cochlea, Presbycusis. Notable publications include 'The nuclear transcription factor FoxG1 affects the sensitivity of mimetic aging hair cells to inflammation by regulating autophagy pathways', 'FOXG1 promotes aging inner ear hair cell survival through activation of the autophagy pathway', and 'Down regulated connexin26 at different postnatal stage displayed different types of cellular degeneration and formation of organ of Corti'.
h-index
citations
506
works
10
NIH funding
primary concept
email

Recent publications

Abnormal Innervation, Demyelination, and Degeneration of Spiral Ganglion Neurons as Well as Disruption of Heminodes are Involved in the Onset of Deafness in Cx26 Null Mice
Neuroscience Bulletin 2024cited by 12position: middledoi
The protective effects of systemic dexamethasone on sensory epithelial damage and hearing loss in targeted Cx26-null mice
Cell Death and Disease 2022cited by 28position: middledoi
F-Actin Dysplasia Involved in Organ of Corti Deformity in Gjb2 Knockdown Mouse Model
Frontiers in Molecular Neuroscience 2022cited by 16position: middledoi
FOXG1 promotes aging inner ear hair cell survival through activation of the autophagy pathway
Autophagy 2021cited by 128position: middledoi
FOXG1 promotes aging inner ear hair cell survival through activation of the autophagy pathway
Figshare 2021cited by 3position: middledoi
The nuclear transcription factor FoxG1 affects the sensitivity of mimetic aging hair cells to inflammation by regulating autophagy pathways
Redox Biology 2019cited by 166position: middledoi
The spatial distribution pattern of Connexin26 expression in supporting cells and its role in outer hair cell survival
Cell Death and Disease 2018cited by 31position: firstdoi
Reduced Connexin26 in the Mature Cochlea Increases Susceptibility to Noise-Induced Hearing Loss in Mice
International Journal of Molecular Sciences 2016cited by 39position: middledoi
Down regulated connexin26 at different postnatal stage displayed different types of cellular degeneration and formation of organ of Corti
Biochemical and Biophysical Research Communications 2014cited by 45position: firstdoi
Reduced expression of Connexin26 and its DNA promoter hypermethylation in the inner ear of mimetic aging rats induced by d-galactose
Biochemical and Biophysical Research Communications 2014cited by 38position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Yu Sun · China National Offshore Oil Corporation (China)9 papers (2014–2024)Weijia Kong · Wuhan Union Hospital8 papers (2014–2022)Le Xie · Union Hospital5 papers (2016–2024)Xi Lin · Huazhong University of Science and Technology4 papers (2014–2018)Zuhong He · Nantong University4 papers (2018–2021)Yujuan Hu · Ministry of Education of the People's Republic of China3 papers (2019–2021)Kai Xu · Nanchang University3 papers (2018–2022)Shengyu Zou · Union Hospital3 papers (2019–2021)Haiying Sun · Union Hospital3 papers (2019–2021)Xia Wu · Beijing Institute of Technology3 papers (2016–2021)Xueyan Zhao · University of Alabama at Birmingham3 papers (2019–2021)Xiaoxiang Xu · Anhui Medical University3 papers (2019–2021)Renjie Chai · Fudan University3 papers (2019–2021)Fu-ling Liao · Union Hospital3 papers (2019–2021)Yue Qiu · Union Hospital3 papers (2022–2024)Xia Wu · Southeast University2 papers (2014–2021)Yuan Jin · Union Hospital2 papers (2022–2022)Xue Bai · Nanchang University2 papers (2022–2024)Xiaozhou Liu · Union Hospital2 papers (2022–2022)Qiaojun Fang · Nantong University2 papers (2021–2021)
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