Area of research
Cancer Research · Otorhinolaryngology
Research interest
Research focused on Cancer research and Apoptosis, with related work in Metastasis, Breast cancer, Gene silencing. Notable publications include 'MiR-485-3p and miR-485-5p suppress breast cancer cell metastasis by inhibiting PGC-1α expression', 'MiR-128 regulation of glucose metabolism and cell proliferation in triple-negative breast cancer', and 'Long non‐coding RNA AFAP1‐AS1/miR‐320a/RBPJ axis regulates laryngeal carcinoma cell stemness and chemoresistance'.
LncRNA LINC-PINT Inhibits Malignant Behaviors of Laryngeal Squamous Cell Carcinoma Cells via Inhibiting ZEB1
Efficacy of superselective neck dissection (IIA and III) for supraglottic laryngeal cancer with clinically negative neck
Long non‐coding RNA AFAP1‐AS1/miR‐320a/RBPJ axis regulates laryngeal carcinoma cell stemness and chemoresistance
MiR-128 regulation of glucose metabolism and cell proliferation in triple-negative breast cancer
MiR-485-3p and miR-485-5p suppress breast cancer cell metastasis by inhibiting PGC-1α expression
Different expression of sodium–iodide importer (NIS) between lactating breast and thyroid tissues may be due to structural difference of thyroid-stimulating hormone receptor (TSHR)
Effect of EphA7 Silencing on Proliferation, Invasion and Apoptosis in Human Laryngeal Cancer Cell Lines Hep-2 and AMC-HN-8
MicroRNA-9 as a novel prognostic biomarker in human laryngeal squamous cell carcinoma.
PubMed 2014cited by 24position: middle
Lepista sordida polysaccharide induces apoptosis of Hep-2 cancer cells via mitochondrial pathway
Predictive Factors for Different Subgroups of Central Lymph Node Metastasis in Unilateral Papillary Thyroid Carcinoma
Krüppel-like factor 5: a novel biomarker for lymph node metastasis and recurrence in supraglottic squamous cell laryngeal carcinoma
Incidence of level IIB lymph node metastasis in supraglottic laryngeal squamous cell carcinoma with clinically negative neck—A prospective study
Rapamycin inhibits the invasive ability of thyroid cancer cells by down‐regulating the expression of VEGF‐C <i>in vitro</i>