Rong Yin

Tumor-infiltrating B cells participate in shaping the immunosuppressive microenvironment in solid tumors.

Stress granules restrain ferroptosis by sequestering ferritin

Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in EGFR-Mutant Lung Cancer.

Decoding Brain Metastasis With Liquid Biopsy: Biomarkers and Technologies in EGFR-Mutant NSCLC.

Hypoxic niche drives lineage imbalance and early tumorigenesis in EGFR-mutant lung cancer.

Figure S1 from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Figure S3 from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Figure S9 from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Figure S4 from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Figure S6 from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Table S1 from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Figure S10 from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Figure S5 from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Figure S2 from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Figure S8 from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Data from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Figure S7 from Transfer of Damaged Mitochondria from Cancer Cells to Cancer-Associated Fibroblasts Promotes Tyrosine Kinase Inhibitor Tolerance in <i>EGFR</i>-Mutant Lung Cancer

Persist or resist: Immune checkpoint inhibitors in EGFR-mutated NSCLC.

Tumor-derived extracellular vesicle PD-1 promotes tumor immune evasion via disruption of peripheral T cell homeostasis.

Targeting LINC01711 in FAP⁺ cancer-associated fibroblasts overcomes lactate-mediated immunosuppression and enhances anti-PD-1 efficacy in lung adenocarcinoma.

Persistent lineage plasticity driving lung cancer development and progression.

RASON promotes KRAS<sup>G12C</sup>-driven tumor progression and immune evasion in non-small cell lung cancer.

<i>ANKRD11</i> as a potential biomarker for brain metastasis from lung adenocarcinoma via cerebrospinal fluid liquid biopsy.

Massively parallel variant-to-function mapping determines functional regulatory variants of non-small cell lung cancer.

Comprehensive Characterization of Somatic Mutation Timing Reveals the Evolutionary Trajectory of Lung Adenocarcinoma in Chinese Patients.

Comprehensive Characterization of Somatic Mutation Timing Reveals the Evolutionary Trajectory of Lung Adenocarcinoma in Chinese Patients

Supplementary Fig.S9 from Comprehensive Characterization of Somatic Mutation Timing Reveals the Evolutionary Trajectory of Lung Adenocarcinoma in Chinese Patients

Supplementary Table S1 from Comprehensive Characterization of Somatic Mutation Timing Reveals the Evolutionary Trajectory of Lung Adenocarcinoma in Chinese Patients

Supplementary Fig.S1 from Comprehensive Characterization of Somatic Mutation Timing Reveals the Evolutionary Trajectory of Lung Adenocarcinoma in Chinese Patients

Supplementary Fig.S5 from Comprehensive Characterization of Somatic Mutation Timing Reveals the Evolutionary Trajectory of Lung Adenocarcinoma in Chinese Patients

CAREER: Fabrication of Sustainable Hemp Yarns and Textiles Using a Modified Fiber Spinning Process