Area of research
Molecular Biology · Health, Toxicology and Mutagenesis
Research interest
Research interests include Cancer research, Biology, Histone, Carcinogenesis, Carcinogen, and Epigenetics.
Ellagic acid alleviates sepsis-induced intestinal injury by modulating gut microbiota and NF-κB-mediated MLCK/MLC signaling pathway
Targeted inhibition of the HNF1A/SHH axis by triptolide overcomes paclitaxel resistance in non-small cell lung cancer
Polyadenylation of Histone H3.1 mRNA Promotes Cell Transformation by Displacing H3.3 from Gene Regulatory Elements
Downregulation of hedgehog-interacting protein (HHIP) contributes to hexavalent chromium-induced malignant transformation of human bronchial epithelial cells
Molecular and epigenetic mechanisms of Cr(VI)-induced carcinogenesis
Overexpressed miR-200a promotes bladder cancer invasion through direct regulating Dicer/miR-16/JNK2/MMP-2 axis
Role of miR‐31 and SATB2 in arsenic‐induced malignant BEAS‐2B cell transformation
XIAP BIR domain suppresses miR-200a expression and subsequently promotes EGFR protein translation and anchorage-independent growth of bladder cancer cell
Transcription factors and stress response gene alterations in human keratinocytes following Solar Simulated Ultra Violet Radiation
Induction of miR-137 by Isorhapontigenin (ISO) Directly Targets Sp1 Protein Translation and Mediates Its Anticancer Activity Both<i>In Vitro</i>and<i>In Vivo</i>
Oral Chromium Exposure and Toxicity
Tungsten-induced carcinogenesis in human bronchial epithelial cells
Gene expression and pathway analysis of human hepatocellular carcinoma cells treated with cadmium
Arsenic Induces Polyadenylation of Canonical Histone mRNA by Down-regulating Stem-Loop-binding Protein Gene Expression
Nickel and Epigenetic Gene Silencing
Associations between Arsenic Exposure and Global Posttranslational Histone Modifications among Adults in Bangladesh
Gene expression changes in human lung cells exposed to arsenic, chromium, nickel or vanadium indicate the first steps in cancer
The effect of exposure to carcinogenic metals on histone tail modifications and gene expression in human subjects