Area of research
Epidemiology · Cancer Research
Research interest
Research focused on Autophagy and PI3K/AKT/mTOR pathway, with related work in Protein kinase B, Sorafenib, Apoptosis. Notable publications include 'Quercetin prevents hepatic fibrosis by inhibiting hepatic stellate cell activation and reducing autophagy via the TGF-β1/Smads and PI3K/Akt pathways', 'Subepicardial endothelial cells invade the embryonic ventricle wall to form coronary arteries', and 'Genistein suppresses aerobic glycolysis and induces hepatocellular carcinoma cell death'.
An inkjet-printable organic electrochemical transistor array with differentiated ion dynamics for sweat fingerprint identification
Ductal or Ngn3+ cells do not contribute to adult pancreatic islet beta-cell neogenesis in homeostasis
Integrated analyses of m <sup>1</sup> A regulator-mediated modification patterns in tumor microenvironment-infiltrating immune cells in colon cancer
Quercetin prevents hepatic fibrosis by inhibiting hepatic stellate cell activation and reducing autophagy via the TGF-β1/Smads and PI3K/Akt pathways
Genistein suppresses aerobic glycolysis and induces hepatocellular carcinoma cell death
By inhibiting PFKFB3, aspirin overcomes sorafenib resistance in hepatocellular carcinoma
The protective effects of shikonin on hepatic ischemia/reperfusion injury are mediated by the activation of the PI3K/Akt pathway
The long noncoding RNA TUG1 acts as a competing endogenous RNA to regulate the Hedgehog pathway by targeting miR-132 in hepatocellular carcinoma
Quercetin Pretreatment Attenuates Hepatic Ischemia Reperfusion-Induced Apoptosis and Autophagy by Inhibiting ERK/NF-<i>κ</i>B Pathway
Pretreatment with propylene glycol alginate sodium sulfate ameliorated concanavalin A-induced liver injury by regulating the PI3K/Akt pathway in mice
The natural product fucoidan ameliorates hepatic ischemia–reperfusion injury in mice
Mfsd2a+ hepatocytes repopulate the liver during injury and regeneration
Subepicardial endothelial cells invade the embryonic ventricle wall to form coronary arteries