Area of research
Cellular and Molecular Neuroscience · Physiology
Research interest
Research interests include Pain Mechanisms and Treatments, Neuroscience and Neuropharmacology Research, Neuropeptides and Animal Physiology, and Ion channel regulation and function.
Self-assembling natural flavonoid nanomedicines for alveolar macrophage reprogramming by restoring mitochondrial function in acute lung injury therapy
Mitochondrial dysfunction reveals H2S-mediated synaptic sulfhydration as a potential mechanism for autism-associated social defects
BIN1 Interacts with Tau Fragments to Inhibit TrkB Signaling Endosome Recycling in a Mouse Model of Alzheimer’s Disease
Corrigendum to “Self-assembling Natural Flavonoid Nanomedicines for Alveolar Macrophage Reprogramming by Restoring Mitochondrial Function in Acute Lung Injury Therapy”[Chem. Eng. J. 506 (2025) 160171/CEJ 160171]
Dorsal raphe nucleus–hippocampus serotonergic circuit underlies the depressive and cognitive impairments in 5×FAD male mice
Quiescent Adult Neural Stem Cells: Developmental Origin and Regulatory Mechanisms
Development of an α-synuclein positron emission tomography tracer for imaging synucleinopathies
Characteristics of traumatic brain injury models: from macroscopic blood flow changes to microscopic mitochondrial changes
Glutamatergic Neurons in the Amygdala Are Involved in Paclitaxel-Induced Pain and Anxiety
IRES-mediated Wnt2 translation in apoptotic neurons triggers astrocyte dedifferentiation
Spinal CCL2 Promotes Pain Sensitization by Rapid Enhancement of NMDA-Induced Currents Through the ERK-GluN2B Pathway in Mouse Lamina II Neurons
Anterior cingulate cortex dysfunction underlies social deficits in Shank3 mutant mice
Deficiency in BDNF/TrkB Neurotrophic Activity Stimulates δ-Secretase by Upregulating C/EBPβ in Alzheimer’s Disease
Delta-secretase-cleaved Tau antagonizes TrkB neurotrophic signalings, mediating Alzheimer’s disease pathologies
Astroglial Kir4.1 in the lateral habenula drives neuronal bursts in depression
Striatopallidal dysfunction underlies repetitive behavior in Shank3-deficient model of autism
Spinal CCL2 Promotes Central Sensitization, Long-Term Potentiation, and Inflammatory Pain via CCR2: Further Insights into Molecular, Synaptic, and Cellular Mechanisms