Area of research
Molecular Biology · Cancer Research
Research interest
Research focused on Protein kinase B and Apoptosis, with related work in Demethylase, AMPK, PI3K/AKT/mTOR pathway. Notable publications include 'N6-Methyladenosine co-transcriptionally directs the demethylation of histone H3K9me2', 'Discrete mechanisms of mTOR and cell cycle regulation by AMPK agonists independent of AMPK', and 'Myeloid Malignancies with Chromosome 5q Deletions Acquire a Dependency on an Intrachromosomal NF-κB Gene Network'.
Blueberry anthocyanins ameliorate arsenic-induced cognitive impairment in rats: mitigating mitochondrial damage and dysregulation
Evaluating a new obesity indicator for stroke risk prediction: comparative cohort analysis in rural settings of two nations
Impaired learning and memory in male mice induced by sodium arsenite was associated with MMP-2/MMP-9-mediated blood-brain barrier disruption and neuronal apoptosis
Arsenic-Induced, Mitochondria-Mediated Apoptosis Is Associated with Decreased Peroxisome Proliferator-Activated Receptor γ Coactivator α in Rat Brains
The disruption of blood-brain barrier induced by long-term arsenic exposure is associated with the increase of MMP-9 and MMP-2: The characteristics are similar to those caused by senescence
Environment and chronic disease in rural areas of Heilongjiang, China (ECDRAHC)
Arsenic exposure elevated ROS promotes energy metabolic reprogramming with enhanced AKT-dependent HK2 expression
Malignant growth of arsenic-transformed cells depends on activated Akt induced by reactive oxygen species
N6-Methyladenosine co-transcriptionally directs the demethylation of histone H3K9me2
sKlotho is associated with the severity of brick tea-type skeletal fluorosis in China
Pigment epithelium-derived factor (PEDF) ameliorates arsenic-induced vascular endothelial dysfunction in rats and toxicity in endothelial EA.hy926 cells
Vitamin E derivative-based multifunctional nanoemulsions for overcoming multidrug resistance in cancer
Role of PTEN-Akt-CREB Signaling Pathway in Nervous System impairment of Rats with Chronic Arsenite Exposure
Neuroglobin Plays a Protective Role in Arsenite-Induced Cytotoxicity by Inhibition of Cdc42 and Rac1GTPases in Rat Cerebellar Granule Neurons
Discrete mechanisms of mTOR and cell cycle regulation by AMPK agonists independent of AMPK
Myeloid Malignancies with Chromosome 5q Deletions Acquire a Dependency on an Intrachromosomal NF-κB Gene Network
Neuroglobin Involvement in the Course of Arsenic Toxicity in Rat Cerebellar Granule Neurons
p38 and Extracellular Signal‐Regulated Kinases Activations have Opposite Effects on Primary‐Cultured Rat Cerebellar Granule Neurons Exposed to Sodium Arsenite