Area of research
Molecular Biology · Nephrology
Research interest
Research focused on Klotho and Gene knockdown, with related work in Calcification, Vascular smooth muscle, Downregulation and upregulation. Notable publications include 'Klotho/FGF23 axis mediates high phosphate‐induced vascular calcification in vascular smooth muscle cells via Wnt7b/β‐catenin pathway', 'Long noncoding RNA NEAT1 sponges miR-129 to modulate renal fibrosis by regulation of collagen type I', and 'Overexpression of HOTAIR attenuates Pi-induced vascular calcification by inhibiting Wnt/β-catenin through regulating miR-126/Klotho/SIRT1 axis'.
Sepsis-induced ALI/ARDS beyond supportive care: molecular mechanisms and cutting-edge therapies
Angiotensin-(1–7) Mitigates Progression from Acute Kidney Injury to Chronic Kidney Disease Via Renin-Angiotensin System Modulation in a Murine Model
Severe asthma beyond bronchodilators: Emerging therapeutic approaches
Relationship of serum calcium concentration with chronic kidney disease and mortality in type 2 diabetes mellitus patients: evidence from the NHANES 1999–2018
Pegmolesatide for the treatment of anemia in patients undergoing dialysis: a randomized clinical trial
Allograft inflammatory factor 1 is a potential diagnostic, immunological, and prognostic biomarker in pan-cancer
Analysis of the impact of peritoneal dialysis catheter tail‐end design on catheter‐related complications
Influence of sodium thiosulfate on coronary artery calcification of patients on dialysis: a meta-analysis
LncRNA NEAT1 accelerates renal fibrosis progression via targeting miR-31 and modulating RhoA/ROCK signal pathway
LncRNA Dlx6os1 Accelerates Diabetic Nephropathy Progression by Epigenetically Repressing SOX6 via Recruiting EZH2
Overexpression of HOTAIR attenuates Pi-induced vascular calcification by inhibiting Wnt/β-catenin through regulating miR-126/Klotho/SIRT1 axis
Overexpression of SP1 restores autophagy to alleviate acute renal injury induced by ischemia-reperfusion through the miR-205/PTEN/Akt pathway
The Clinical Value of Klotho and FGF23 in Cardiac Valve Calcification Among Patients with Chronic Kidney Disease
Long noncoding RNA NEAT1 sponges miR-129 to modulate renal fibrosis by regulation of collagen type I
LncRNA-SNHG29 inhibits vascular smooth muscle cell calcification by downregulating miR-200b-3p to activate the α-Klotho/FGFR1/FGF23 axis
Factors Associated with a Large Decline in Renal Function or Progression to Renal Insufficiency in Hospitalized Atrial Fibrillation Patients with Early-Stage CKD
Klotho/FGF23 axis mediates high phosphate‐induced vascular calcification in vascular smooth muscle cells via Wnt7b/β‐catenin pathway