Area of research
Immunology · Surgery
Research interest
Research interests include Immune Cell Function and Interaction, T-cell and B-cell Immunology, Organ Transplantation Techniques and Outcomes, and Immunotherapy and Immune Responses.
Peripheral nervous system microglia-like cells regulate neuronal soma size throughout evolution
FOXM1 is critical for the fitness recovery of chromosomally unstable cells
A Biomimetic Aggregation‐Induced Emission Photosensitizer with Antigen‐Presenting and Hitchhiking Function for Lipid Droplet Targeted Photodynamic Immunotherapy
Intestinal microbiota-derived short-chain fatty acids regulation of immune cell IL-22 production and gut immunity
Mechanisms regulating PD-L1 expression on tumor and immune cells
A biologic scaffold–associated type 2 immune microenvironment inhibits tumor formation and synergizes with checkpoint immunotherapy
Nemo-like Kinase Drives Foxp3 Stability and Is Critical for Maintenance of Immune Tolerance by Regulatory T Cells
Chemotherapy induces enrichment of CD47 <sup>+</sup> /CD73 <sup>+</sup> /PDL1 <sup>+</sup> immune evasive triple-negative breast cancer cells
YAP Is Essential for Treg-Mediated Suppression of Antitumor Immunity
The regulation of immune tolerance by FOXP3
TGFβ1-Mediated SMAD3 Enhances PD-1 Expression on Antigen-Specific T Cells in Cancer
Foxp3, Regulatory T Cell, and Autoimmune Diseases
Metabolic control of type 1 regulatory T cell differentiation by AHR and HIF1-α
Treg functional stability and its responsiveness to the microenvironment
STAT3 regulates arginase-I in myeloid-derived suppressor cells from cancer patients
Stabilization of the Transcription Factor Foxp3 by the Deubiquitinase USP7 Increases Treg-Cell-Suppressive Capacity
The Ubiquitin Ligase Stub1 Negatively Modulates Regulatory T Cell Suppressive Activity by Promoting Degradation of the Transcription Factor Foxp3
Metabolic control of the Treg/Th17 axis
An Inherently Bifunctional Subset of Foxp3+ T Helper Cells Is Controlled by the Transcription Factor Eos
Loss of Epigenetic Modification Driven by the Foxp3 Transcription Factor Leads to Regulatory T Cell Insufficiency