Area of research
Nephrology · Clinical Biochemistry
Research interest
Research interests include Chronic Kidney Disease and Diabetes, Dialysis and Renal Disease Management, Renal Diseases and Glomerulopathies, and Advanced Glycation End Products research.
Empagliflozin attenuates renal damage in diabetic nephropathy by modulating mitochondrial quality control via Prdx3-PINK1 pathway
Mechanism of tripterygium glycoside in reducing proteinuria in diabetic nephropathy rats through RhoA/ROCK1 and Wnt1/β-catenin signaling pathways
Honokiol attenuates diabetic nephropathy by targeting SIRT3 to suppress mitochondrial ROS-induced pyroptosis
Causal relationship and shared genetic pathways between diabetic kidney disease and cognitive impairment: a Mendelian randomization study
The efficacy of percutaneous transluminal angioplasty and arteriovenous fistula reconstruction for immature arteriovenous fistula
Insights into the effects of Al-ion implantation temperature on material properties of 4H-SiC
Effect and mechanism of TFEB on pyroptosis in HK-2 cells induced by high glucose
Pyridoxamine alleviates high glucose induced fibrosis in renal tubular epithelial cell by inhibiting the activity of TGF-β1/Smad3 signaling pathway
Resveratrol Reduces Oxidative Stress and Apoptosis in Podocytes via Sir2-Related Enzymes, Sirtuins1 (SIRT1)/Peroxisome Proliferator-Activated Receptor γ Co-Activator 1α (PGC-1α) Axis
Protective effects of pyrroloquinoline quinone against oxidative stress-induced cellular senescence and inflammation in human renal tubular epithelial cells via Keap1/Nrf2 signaling pathway
Effects of transcription factor EB on oxidative stress and apoptosis induced by high glucose in podocytes
Pyridoxamine Treatment of HK-2 Human Proximal Tubular Epithelial Cells Reduces Oxidative Stress and the Inhibition of Autophagy Induced by High Glucose Levels
Resveratrol ameliorates podocyte damage in diabetic mice via SIRT1/PGC‐1α mediated attenuation of mitochondrial oxidative stress
Pyrroloquinoline quinine protects HK-2 cells against high glucose-induced oxidative stress and apoptosis through Sirt3 and PI3K/Akt/FoxO3a signaling pathway
Genistein and Myd88 Activate Autophagy in High Glucose-Induced Renal Podocytes In Vitro
Salidroside Reduces High-Glucose-Induced Podocyte Apoptosis and Oxidative Stress via Upregulating Heme Oxygenase-1 (HO-1) Expression
Matrix metalloproteinase 12 modulates high-fat-diet induced glomerular fibrogenesis and inflammation in a mouse model of obesity
Role of Sirtuin3 in high glucose-induced apoptosis in renal tubular epithelial cells
The Notch pathway mediates the angiotensin II-induced synthesis of extracellular matrix components in podocytes
Mechanism of Genistein in Autophagy of High Glucose Ambient Mice Podocyte
Benazepril affects integrin-linked kinase and smooth muscle α-actin expression in diabetic rat glomerulus and cultured mesangial cells
Gremlin aggravates hyperglycemia‐induced podocyte injury by a TGFβ/smad dependent signaling pathway
Gremlin induces cell proliferation and extra cellular matrix accumulation in mouse mesangial cells exposed to high glucose via the ERK1/2 pathway