Area of research
Environmental Chemistry · Health, Toxicology and Mutagenesis
Research interest
Research interests include Oxidative stress, Apoptosis, Curcumin, Arsenic trioxide, Pyroptosis, and Chemistry.
Protective effect of curcumin against endoplasmic reticulum stress and lipid metabolism disorders in AFB1-intoxicated duck liver
Curcumin attenuates aflatoxin B1-induced ileum injury in ducks by inhibiting NLRP3 inflammasome and regulating TLR4/NF-κB signaling pathway
Co-exposure of arsenic and polystyrene-nanoplastics induced kidney injury by disrupting mitochondrial homeostasis and mtROS-mediated ferritinophagy and ferroptosis
Arsenic and polystyrene‐nano plastics co‐exposure induced testicular toxicity: Triggers oxidative stress and promotes apoptosis and inflammation in mice
Arsenic induced neurotoxicity in the brain of ducks: The potential involvement of the gut-brain axis
Combined effect of arsenic and polystyrene-nanoplastics at environmentally relevant concentrations in mice liver: Activation of apoptosis, pyroptosis and excessive autophagy
Curcumin attenuates AFB1-induced duck liver injury by inhibiting oxidative stress and lysosomal damage
Curcumin activates the Nrf2 Pathway to alleviate AFB1-induced immunosuppression in the spleen of ducklings
Curcumin Alleviates Arsenic Trioxide–Induced Inflammation and Pyroptosis via the NF-κB/NLRP3 Signaling Pathway in the Hypothalamus of Ducks
Arsenic Trioxide Triggers Mitochondrial Dysfunction, Oxidative Stress, and Apoptosis via Nrf 2/Caspase 3 Signaling Pathway in Heart of Ducks
Correction to: Arsenic Trioxide Triggers Mitochondrial Dysfunction, Oxidative Stress, and Apoptosis via Nrf 2/Caspase 3 Signaling Pathway in Heart of Ducks
Evaluation of toxic effects induced by arsenic trioxide or/and antimony on autophagy and apoptosis in testis of adult mice
The neuroprotective effect of curcumin against ATO triggered neurotoxicity through Nrf2 and NF-κB signaling pathway in the brain of ducks