Area of research
Immunology · Immunology and Allergy
Research interest
Research interests include Cell Adhesion Molecules Research, Head and Neck Cancer Studies, Neutrophil, Myeloperoxidase and Oxidative Mechanisms, and Immune Response and Inflammation.
Personalized ctDNA analysis for detection of residual disease and recurrence in surgically treated HNSCC patients
EGFR-mediated local invasiveness and response to Cetuximab in head and neck cancer
Procoagulant platelets promote immune evasion in triple-negative breast cancer
Pro-coagulant platelets promote immune evasion
Prokoagulante Thrombozyten vermitteln die intratumorale Fehlleitung von Immunzellen
A transcriptomic pan-cancer signature for survival prognostication and prediction of immunotherapy response based on endothelial senescence
S2k Guideline for the diagnosis and treatment of mucous membrane pemphigoid
S2k‐Leitlinie zur Diagnostik und Therapie des Schleimhautpemphigoids
uPA‐PAI‐1 heteromerization promotes breast cancer progression by attracting tumorigenic neutrophils
Low kindlin-3 levels in osteoclasts of kindlin-3 hypomorphic mice result in osteopetrosis due to leaky sealing zones
P02.09 Heteromerization of uPA and PAI-1 enforces pro-tumorigenic neutrophil trafficking to malignant tumors in breast cancer <i>via</i> VLDLr-dependent β2 integrin clustering
Priming of Anti-tumor Immune Mechanisms by Radiotherapy Is Augmented by Inhibition of Heat Shock Protein 90
Epithelial-type systemic breast carcinoma cells with a restricted mesenchymal transition are a major source of metastasis
Artery-Associated Sympathetic Innervation Drives Rhythmic Vascular Inflammation of Arteries and Veins
Priming anti-tumor immunity by radiotherapy: Dying tumor cell-derived DAMPs trigger endothelial cell activation and recruitment of myeloid cells
Vitronectin promotes the vascularization of porous polyethylene biomaterials
Cytotoxicity of crystals involves RIPK3-MLKL-mediated necroptosis
Synchronized renal tubular cell death involves ferroptosis
Endogenous and exogenous pentraxin-3 limits postischemic acute and chronic kidney injury
Histones from Dying Renal Cells Aggravate Kidney Injury via TLR2 and TLR4