Area of research
Emergency Medical Services · Physiology
Research interest
Research focused on Bronchoalveolar lavage and COPD, with related work in Inflammation, Lipopolysaccharide, Proinflammatory cytokine. Notable publications include 'Jianpiyifei II Granules Suppress Apoptosis of Bronchial Epithelial Cells in Chronic Obstructive Pulmonary Disease via Inhibition of the Reactive Oxygen Species-Endoplasmic...', 'Molecular mechanism of Jianpiyifei II granules in the treatment of chronic obstructive pulmonary disease: Network pharmacology analysis, molecular docking, and experimental...', and 'Luteolin alleviates airway remodeling in asthma by inhibiting the epithelial-mesenchymal transition via β-catenin regulation'.
Correction: Short-term cigarette smoke exposure aggravates oxidative stress and airway inflammation induced by lipopolysaccharides
Investigation of the effect and mechanisms of moxa smoke in the treatment of Influenza A Virus (IAV) infection
Efficacy of Kegan Liyan oral liquid vs. Lianhuaqingwen capsules for patients with mild COVID-19: a double-blinded, randomized, controlled, non-inferiority trial
Molecular mechanism of Jianpiyifei II granules in the treatment of chronic obstructive pulmonary disease: Network pharmacology analysis, molecular docking, and experimental assessment
Luteolin alleviates airway remodeling in asthma by inhibiting the epithelial-mesenchymal transition via β-catenin regulation
Inhibition of Inflammation and Regulation of AQPs/ENaCs/Na+-K+-ATPase Mediated Alveolar Fluid Transport by Total Flavonoids Extracted From Nervilia fordii in Lipopolysaccharide-induced Acute Lung Injury
The traditional herbal formulation, <i>Jianpiyifei II</i>, reduces pulmonary inflammation induced by influenza A virus and cigarette smoke in mice
Jianpiyifei II Granules Suppress Apoptosis of Bronchial Epithelial Cells in Chronic Obstructive Pulmonary Disease via Inhibition of the Reactive Oxygen Species-Endoplasmic Reticulum Stress-Ca2+ Signaling Pathway
Protective Effect of Jianpiyifei II Granule against Chronic Obstructive Pulmonary Disease via NF‐<i>κ</i>B Signaling Pathway