Area of research
Biochemistry · Physiology
Research interest
Research focused on SIRT3 and Ubiquitin ligase, with related work in Ubiquitin, Mitochondrion, Cell biology. Notable publications include 'Exogenous H2S Promoted USP8 Sulfhydration to Regulate Mitophagy in the Hearts of db/db Mice', 'Exogenous H2S switches cardiac energy substrate metabolism by regulating SIRT3 expression in db/db mice', and 'Exogenous H 2 S reduces the acetylation levels of mitochondrial respiratory enzymes via regulating the NAD + -SIRT3 pathway in cardiac tissues of db/db mice'.
Intensive Blood Pressure Control in Older Patients With Hypertension
Translocation of SIRT6 promotes glycolysis reprogramming to exacerbate diabetic angiopathy
Hydrogen sulfide improves endothelial barrier function by modulating the ubiquitination degradation of KLF4 through TRAF7 S-sulfhydration in diabetic aorta
Qiliqiangxin Alleviates Imbalance of Inflammatory Cytokines in Patients with Dilated Cardiomyopathy: A Randomized Controlled Trial
Exogenous hydrogen sulfide enhances myogenic differentiation of C2C12 myoblasts under high palmitate stress
Hydrogen sulfide regulates SERCA2a SUMOylation by S-Sulfhydration of SENP1 to ameliorate cardiac systole-diastole function in diabetic cardiomyopathy
The Influence of baseline glycemic status on the effects of intensive blood pressure lowering: Results from the STEP randomized trial
PDZK1 improves ventricular remodeling in hypertensive rats by regulating the stability of the Mas receptor
Hydrogen Sulfide Regulates SERCA2a Ubiquitylation via Muscle RING Finger-1 S-Sulfhydration to Affect Cardiac Contractility in db/db Mice
Exogenous H2S Promoted USP8 Sulfhydration to Regulate Mitophagy in the Hearts of db/db Mice
Exogenous H2S Induces Hrd1 S-sulfhydration and Prevents CD36 Translocation via VAMP3 Ubiquitylation in Diabetic Hearts
Hydrogen sulphide ameliorating skeletal muscle atrophy in db/db mice via Muscle RING finger 1 S‐sulfhydration
Exogenous H<sub>2</sub>S reduces the acetylation levels of mitochondrial respiratory enzymes via regulating the NAD<sup>+</sup>-SIRT3 pathway in cardiac tissues of <i>db/db</i> mice
Hydrogen sulfide regulates muscle RING finger‐1 protein S‐sulfhydration at Cys<sup>44</sup> to prevent cardiac structural damage in diabetic cardiomyopathy
MiR-146a down-regulates inflammatory response by targeting TLR3 and TRAF6 in Coxsackievirus B infection
Exogenous H2S switches cardiac energy substrate metabolism by regulating SIRT3 expression in db/db mice
Nox4-dependent ROS production is involved in CVB3-induced myocardial apoptosis
GW26-e1516 The Effects of Nox4 on Myocardial Apoptosis in CoxsackievirusB3 Induced Myocarditis
Cyclosporin A induces apoptosis in H9c2 cardiomyoblast cells through calcium-sensing receptor-mediated activation of the ERK MAPK and p38 MAPK pathways
Knockdown of dishevelled-1 attenuates cyclosporine A-induced apoptosis in H9c2 cardiomyoblast cells
Rho kinase in cardiovascular diseases and applications for Rho kinase inhibition: Current progress
Chinese Heart Journal 2012cited by 0position: first