Area of research
Cardiology and Cardiovascular Medicine · Radiology, Nuclear Medicine and Imaging
Research interest
Research interests include Cardiomyopathy and Myosin Studies, Cardiovascular Function and Risk Factors, Cardiovascular Effects of Exercise, and Cardiac Imaging and Diagnostics.
Abstract Fri009: Connexin-43 Gene Therapy Targets Desmosomal Gene Expression To Rescue Cardiac End-Stage Deficits in Mouse and Human Models of Arrhythmogenic Cardiomyopathy
Plakophilin 2 gene therapy prevents and rescues arrhythmogenic right ventricular cardiomyopathy in a mouse model harboring patient genetics
Constitutive protein kinase G activation exacerbates stress‐induced cardiomyopathy
Abstract P449: Connexin43 As A Therapeutic For Arrhythmogenic Right Ventricular Cardiomyopathy
Systemic AAV9.LAMP2B injection reverses metabolic and physiologic multiorgan dysfunction in a murine model of Danon disease
Nexilin Is a New Component of Junctional Membrane Complexes Required for Cardiac T-Tubule Formation
Oxidized phospholipids are proinflammatory and proatherogenic in hypercholesterolaemic mice
Reduction of myocardial ischaemia–reperfusion injury by inactivating oxidized phospholipids
Pericytes of Multiple Organs Do Not Behave as Mesenchymal Stem Cells In Vivo
Loss-of-function mutations in co-chaperone BAG3 destabilize small HSPs and cause cardiomyopathy
Impaired mitophagy facilitates mitochondrial damage in Danon disease
Adipocyte-specific loss of PPARγ attenuates cardiac hypertrophy
MLP and CARP are linked to chronic PKCα signalling in dilated cardiomyopathy
Desmosomal junctions are necessary for adult sinus node function
Mitochondrial Reprogramming Induced by CaMKIIδ Mediates Hypertrophy Decompensation
Resident fibroblast lineages mediate pressure overload–induced cardiac fibrosis
The Muscle Ankyrin Repeat Proteins CARP, Ankrd2, and DARP Are Not Essential for Normal Cardiac Development and Function at Basal Conditions and in Response to Pressure Overload
Ca <sup>2+</sup> /Calmodulin-Dependent Protein Kinase II δ Mediates Myocardial Ischemia/Reperfusion Injury Through Nuclear Factor-κB
Connexin defects underlie arrhythmogenic right ventricular cardiomyopathy in a novel mouse model
Akt regulates L-type Ca2+ channel activity by modulating Cavα1 protein stability
Mouse and computational models link Mlc2v dephosphorylation to altered myosin kinetics in early cardiac disease
Talin1 Has Unique Expression versus Talin 2 in the Heart and Modifies the Hypertrophic Response to Pressure Overload