Area of research
Physiology · Molecular Biology
Research interest
Research focused on Skeletal muscle and Duchenne muscular dystrophy, with related work in Bioenergetics, Oxidative phosphorylation, Dystrophin. Notable publications include 'Early myopathy in Duchenne muscular dystrophy is associated with elevated mitochondrial H 2 O 2 emission during impaired oxidative phosphorylation', 'Altered mitochondrial bioenergetics and ultrastructure in the skeletal muscle of young adults with type 1 diabetes', and 'Mitochondrial Energetics in Skeletal Muscle Are Associated With Leg Power and Cardiorespiratory Fitness in the Study of Muscle, Mobility and Aging'.
Skeletal Muscle Composition, Power, and Mitochondrial Energetics in Older Men and Women With Knee Osteoarthritis
Mitohormesis during advanced stages of Duchenne muscular dystrophy reveals a redox-sensitive creatine pathway that can be enhanced by the mitochondrial-targeting peptide SBT-20
Disrupted cardiac bioenergetics linked to oxidized mitochondrial creatine kinase are rescued by the mitochondrial-targeting peptide SBT-20 in the D2.mdx model of Duchenne muscular dystrophy
Mitochondrial Energetics in Skeletal Muscle Are Associated With Leg Power and Cardiorespiratory Fitness in the Study of Muscle, Mobility and Aging
State of Knowledge on Molecular Adaptations to Exercise in Humans: Historical Perspectives and Future Directions
Mitochondrial bioenergetic dysfunction in the D2.mdx model of Duchenne muscular dystrophy is associated with microtubule disorganization in skeletal muscle
Correction to: Altered mitochondrial bioenergetics and ultrastructure in the skeletal muscle of young adults with type 1 diabetes
Early myopathy in Duchenne muscular dystrophy is associated with elevated mitochondrial H<sub>2</sub>O<sub>2</sub> emission during impaired oxidative phosphorylation
Impairments in left ventricular mitochondrial bioenergetics precede overt cardiac dysfunction and remodelling in Duchenne muscular dystrophy
Sexual dimorphism in human skeletal muscle mitochondrial bioenergetics in response to type 1 diabetes
Altered skeletal muscle microtubule-mitochondrial VDAC2 binding is related to bioenergetic impairments after paclitaxel but not vinblastine chemotherapies
Mitochondrial Dysfunction and Disorganized Microtubules in Duchenne Muscular Dystrophy are Not Related to Altered α Tubulin‐Voltage Dependent Anion Channel (VDAC) 2 Interactions
Advanced Stages of Duchenne Muscular Dystrophy Exhibit Mitochondrial Bioenergetic Impairments Linked Specifically to Creatine‐Dependent Energy Exchange
Altered Skeletal Muscle Microtubule‐Mitochondrial Voltage Gated‐Dependent Anion Channel (VDAC) 2 Binding is Related to Bioenergetic Impairments after Paclitaxel but not Vinblastine Chemotherapies
Altered mitochondrial bioenergetics and ultrastructure in the skeletal muscle of young adults with type 1 diabetes
Endothelial-specific FoxO1 depletion prevents obesity-related disorders by increasing vascular metabolism and growth
Holo-lipocalin-2–derived siderophores increase mitochondrial ROS and impair oxidative phosphorylation in rat cardiomyocytes
Author response: Endothelial-specific FoxO1 depletion prevents obesity-related disorders by increasing vascular metabolism and growth
Mitochondrial‐Targeted Peptide SBT‐20 Improves Mitochondrial Bioenergetics In Duchenne Muscular Dystrophy In a Mitochondrial Creatine Kinase Dependent Manner
Microtubule‐Targeting Chemotherapy Causes Mitochondrial Dysfunction in Heart and Skeletal Muscle
Reduced ATGL-mediated lipolysis attenuates β-adrenergic-induced AMPK signaling, but not the induction of PKA-targeted genes, in adipocytes and adipose tissue
Mitochondrial Bioenergetics and Fiber Type Assessments in Microbiopsy vs. Bergstrom Percutaneous Sampling of Human Skeletal Muscle