Area of research
Molecular Biology · Immunology
Research interest
Research focused on Neutrophil extracellular traps and Progenitor cell, with related work in Stem cell, Diabetes mellitus, Neutrophil elastase. Notable publications include 'NETosis Delays Diabetic Wound Healing in Mice and Humans', 'NETosis is induced by high glucose and associated with type 2 diabetes', and 'The antidiabetic drug metformin blunts NETosis in vitro and reduces circulating NETosis biomarkers in vivo'.
Diabetes mellitus impairs circulating proangiogenic granulocytes
Diabetes-Associated Myelopoiesis Drives Stem Cell Mobilopathy Through an OSM-p66Shc Signaling Pathway
The antidiabetic drug metformin blunts NETosis in vitro and reduces circulating NETosis biomarkers in vivo
NETosis Delays Diabetic Wound Healing in Mice and Humans
Bone Marrow Macrophages Contribute to Diabetic Stem Cell Mobilopathy by Producing Oncostatin M
Acute Effects of Linagliptin on Progenitor Cells, Monocyte Phenotypes, and Soluble Mediators in Type 2 Diabetes
A perspective on NETosis in diabetes and cardiometabolic disorders
Diabetes Limits Stem Cell Mobilization Following G-CSF but Not Plerixafor
p66Shc deletion or deficiency protects from obesity but not metabolic dysfunction in mice and humans
NETosis is induced by high glucose and associated with type 2 diabetes
The dipeptidyl peptidase-4 inhibitor Saxagliptin improves function of circulating pro-angiogenic cells from type 2 diabetic patients
Diabetes Causes Bone Marrow Autonomic Neuropathy and Impairs Stem Cell Mobilization via Dysregulated <i>p66Shc</i> and <i>Sirt1</i>
Myeloid calcifying cells promote atherosclerotic calcification via paracrine activity and allograft inflammatory factor-1 overexpression
Endothelial progenitor cells in diabetes mellitus
Stem cell compartmentalization in diabetes and high cardiovascular risk reveals the role of DPP-4 in diabetic stem cell mobilopathy
Procalcific Phenotypic Drift of Circulating Progenitor Cells in Type 2 Diabetes with Coronary Artery Disease