Area of research
Physiology · Immunology
Research interest
Research interests include Nitric Oxide and Endothelin Effects, Neutrophil, Myeloperoxidase and Oxidative Mechanisms, Redox biology and oxidative stress, and Endoplasmic Reticulum Stress and Disease.
NRF2 activation in the heart induces glucose metabolic reprogramming and reduces cardiac dysfunction via upregulation of the pentose phosphate pathway
Human blood vessel organoids reveal a critical role for CTGF in maintaining microvascular integrity
The regulation of cardiac intermediary metabolism by NADPH oxidases
Pkm2 Regulates Cardiomyocyte Cell Cycle and Promotes Cardiac Regeneration
Nox4 regulates InsP <sub>3</sub> receptor‐dependent Ca <sup>2+</sup> release into mitochondria to promote cell survival
Fibroblast Nox2 (NADPH Oxidase-2) Regulates ANG II (Angiotensin II)–Induced Vascular Remodeling and Hypertension via Paracrine Signaling to Vascular Smooth Muscle Cells
Blood Pressure–Lowering by the Antioxidant Resveratrol Is Counterintuitively Mediated by Oxidation of cGMP-Dependent Protein Kinase
Klotho regulation by albuminuria is dependent on ATF3 and endoplasmic reticulum stress
Reactive oxygen species regulate axonal regeneration through the release of exosomal NADPH oxidase 2 complexes into injured axons
Publisher Correction: Reactive oxygen species regulate axonal regeneration through the release of exosomal NADPH oxidase 2 complexes into injured axons
Distinct Regulatory Effects of Myeloid Cell and Endothelial Cell NAPDH Oxidase 2 on Blood Pressure
Nox4 reprograms cardiac substrate metabolism via protein O-GlcNAcylation to enhance stress adaptation
Hypoxia induces heart regeneration in adult mice
Targeted redox inhibition of protein phosphatase 1 by Nox4 regulates <scp>eIF</scp> 2α‐mediated stress signaling
Redox signaling in the cardiomyocyte: From physiology to failure
Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction
Contractile Function During Angiotensin-II Activation
Transcriptional Regulation of Cystathionine-γ-Lyase in Endothelial Cells by NADPH Oxidase 4-Dependent Signaling
The Oxygen-Rich Postnatal Environment Induces Cardiomyocyte Cell-Cycle Arrest through DNA Damage Response
Redox regulation of cardiac hypertrophy
Endoplasmic Reticulum Stress and Nox-Mediated Reactive Oxygen Species Signaling in the Peripheral Vasculature: Potential Role in Hypertension
Pathological Cardiac Hypertrophy Alters Intracellular Targeting of Phosphodiesterase Type 5 From Nitric Oxide Synthase-3 to Natriuretic Peptide Signaling