Area of research
Immunology · Genetics
Research interest
Research focused on Autophagy and ATG16L1, with related work in Immune system, mTORC1, Paneth cell. Notable publications include 'mTORC1 in the Paneth cell niche couples intestinal stem-cell function to calorie intake', '2016 Comprehensive Update of the Banff Working Group on Liver Allograft Pathology: Introduction of Antibody-Mediated Rejection', and 'Interleukin-10 Receptor Signaling in Innate Immune Cells Regulates Mucosal Immune Tolerance and Anti-Inflammatory Macrophage Function'.
102 Immune landscape of adenoid cystic carcinoma using multiplex immunofluorescence and digital pathology
Immunologic Alterations Associated With Oral Delivery of Anti-CD3 (OKT3) Monoclonal Antibodies in Patients With Moderate-to-Severe Ulcerative Colitis
Estrogen-mediated gut microbiome alterations influence sexual dimorphism in metabolic syndrome in mice
<i>C1orf106</i> is a colitis risk gene that regulates stability of epithelial adherens junctions
Predicting the response to CTLA-4 blockade by longitudinal noninvasive monitoring of CD8 T cells
PD-L1 is an activation-independent marker of brown adipocytes
2016 Comprehensive Update of the Banff Working Group on Liver Allograft Pathology: Introduction of Antibody-Mediated Rejection
TMEM258 Is a Component of the Oligosaccharyltransferase Complex Controlling ER Stress and Intestinal Inflammation
The kinase DYRK1A reciprocally regulates the differentiation of Th17 and regulatory T cells
Interleukin-10 Receptor Signaling in Innate Immune Cells Regulates Mucosal Immune Tolerance and Anti-Inflammatory Macrophage Function
Atg16L1 T300A variant decreases selective autophagy resulting in altered cytokine signaling and decreased antibacterial defense
Intestinal alkaline phosphatase prevents metabolic syndrome in mice
Atg16l1 is Required for Autophagy in Intestinal Epithelial Cells and Protection of Mice From Salmonella Infection
mTORC1 in the Paneth cell niche couples intestinal stem-cell function to calorie intake