Area of research
Molecular Biology · Physiology
Research interest
Research focused on Neuroinflammation and CREB, with related work in Basal cell, Cancer research, Neuroscience. Notable publications include 'Sex Differences in Neuropathology and Cognitive Behavior in APP/PS1/tau Triple-Transgenic Mouse Model of Alzheimer’s Disease', 'Lixisenatide reduces amyloid plaques, neurofibrillary tangles and neuroinflammation in an APP/PS1/tau mouse model of Alzheimer's disease', and 'Abnormal circadian locomotor rhythms and Per gene expression in six-month-old triple transgenic mice model of Alzheimer’s disease'.
Effect of defective H2B ubiquitination on the malignancy and repair of DNA double breaks and its significance in lung adenocarcinoma
Epigenetic targeting in oral squamous cell carcinoma: mechanisms, therapies, and translational potential
Hedgehog/Gli2 signaling triggers cell proliferation and metastasis via EMT and wnt/β-catenin pathways in oral squamous cell carcinoma
The role and advance of ubiquitination and deubiquitination in depression pathogenesis and treatment
miR-504 knockout regulates tumor cell proliferation and immune cell infiltration to accelerate oral cancer development
The Chinese hamster as an excellent experimental animal model
Screening and identification of miR-181a-5p in oral squamous cell carcinoma and functional verification in vivo and in vitro
<italic>MCU</italic> knockdown in hippocampal neurons improves memory performance of an Alzheimer’s disease mouse model
<i>miR‐450b</i> promotes cell migration and invasion by inhibiting <i>SERPINB2</i> in oral squamous cell carcinoma
Construction of Fzd6Q152E mice through CRISPR/Cas9 technology and their reproduction and identification
DAla2-GIP-GLU-PAL Protects Against Cognitive Deficits and Pathology in APP/PS1 Mice by Inhibiting Neuroinflammation and Upregulating cAMP/PKA/CREB Signaling Pathways
A dual GLP-1 and Gcg receptor agonist rescues spatial memory and synaptic plasticity in APP/PS1 transgenic mice
Sex Differences in Neuropathology and Cognitive Behavior in APP/PS1/tau Triple-Transgenic Mouse Model of Alzheimer’s Disease
Abnormal circadian locomotor rhythms and Per gene expression in six-month-old triple transgenic mice model of Alzheimer’s disease
A Novel scFv Anti-Aβ Antibody Reduces Pathological Impairments in APP/PS1 Transgenic Mice via Modulation of Inflammatory Cytokines and Aβ-related Enzymes
Lixisenatide reduces amyloid plaques, neurofibrillary tangles and neuroinflammation in an APP/PS1/tau mouse model of Alzheimer's disease
Co-injection of Aβ1-40 and ApoE4 impaired spatial memory and hippocampal long-term potentiation in rats