Area of research
Physiology · Epidemiology
Research interest
Research interests include Adipose Tissue and Metabolism, Liver Disease Diagnosis and Treatment, Pancreatic function and diabetes, and Metabolism, Diabetes, and Cancer.
Isthmin-1 is an adipokine that promotes glucose uptake and improves glucose tolerance and hepatic steatosis
A MicroRNA Linking Human Positive Selection and Metabolic Disorders
Adipsin preserves beta cells in diabetic mice and associates with protection from type 2 diabetes in humans
Anti‐inflammatory effects of oestrogen mediate the sexual dimorphic response to lipid‐induced insulin resistance
Adipose glucocorticoid action influences whole‐body metabolism <i>via</i> modulation of hepatic insulin action
Author Correction: Metformin inhibits gluconeogenesis via a redox-dependent mechanism in vivo
Metformin inhibits gluconeogenesis via a redox-dependent mechanism in vivo
Absence of ANGPTL4 in adipose tissue improves glucose tolerance and attenuates atherogenesis
Genetic Ablation of miR-33 Increases Food Intake, Enhances Adipose Tissue Expansion, and Promotes Obesity and Insulin Resistance
Elevated hepatic expression of H19 long noncoding RNA contributes to diabetic hyperglycemia
Angptl8 antisense oligonucleotide improves adipose lipid metabolism and prevents diet-induced NAFLD and hepatic insulin resistance in rodents
<i>In vivo</i> studies on the mechanism of methylene cyclopropyl acetic acid and methylene cyclopropyl glycine-induced hypoglycemia
UCP1-independent signaling involving SERCA2b-mediated calcium cycling regulates beige fat thermogenesis and systemic glucose homeostasis
Selective Chemical Inhibition of PGC-1α Gluconeogenic Activity Ameliorates Type 2 Diabetes
Absence of Carbohydrate Response Element Binding Protein in Adipocytes Causes Systemic Insulin Resistance and Impairs Glucose Transport
Mechanism by which arylamine <i>N</i> -acetyltransferase 1 ablation causes insulin resistance in mice
A controlled‐release mitochondrial protonophore reverses hypertriglyceridemia, nonalcoholic steatohepatitis, and diabetes in lipodystrophic mice
Adipocyte JAK2 mediates growth hormone–induced hepatic insulin resistance
Adipocyte JAK2 Regulates Hepatic Insulin Sensitivity Independently of Body Composition, Liver Lipid Content, and Hepatic Insulin Signaling
Anti-myostatin antibody increases muscle mass and strength and improves insulin sensitivity in old mice
17α-Estradiol Alleviates Age-related Metabolic and Inflammatory Dysfunction in Male Mice Without Inducing Feminization
Hepatic inositol 1,4,5 trisphosphate receptor type 1 mediates fatty liver
Disruption of Adipose Rab10-Dependent Insulin Signaling Causes Hepatic Insulin Resistance
CD301b + Mononuclear Phagocytes Maintain Positive Energy Balance through Secretion of Resistin-like Molecule Alpha
Reduced intestinal lipid absorption and body weight-independent improvements in insulin sensitivity in high-fat diet-fed <i>Park2</i> knockout mice
Hepatic Acetyl CoA Links Adipose Tissue Inflammation to Hepatic Insulin Resistance and Type 2 Diabetes
Insulin-independent regulation of hepatic triglyceride synthesis by fatty acids
Hepatic insulin resistance and increased hepatic glucose production in mice lacking Fgf21
Reply to Constantin-Teodosiu et al.: Mice with genetic PDH activation are not protected from high-fat diet–induced muscle insulin resistance
Metformin suppresses gluconeogenesis by inhibiting mitochondrial glycerophosphate dehydrogenase