Area of research
Immunology · Cancer Research
Research interest
Research interests include Immune Cell Function and Interaction, T-cell and B-cell Immunology, MicroRNA in disease regulation, and Immunotherapy and Immune Responses.
Single-cell microbiota phenotyping reveals distinct disease and therapy-associated signatures in Crohn’s disease
Clinically used broad-spectrum antibiotics compromise inflammatory monocyte-dependent antibacterial defense in the lung
A type 1 immunity-restricted promoter of the IL−33 receptor gene directs antiviral T-cell responses
IL-33 controls IL-22-dependent antibacterial defense by modulating the microbiota
Distinct tissue niches direct lung immunopathology via CCL18 and CCL21 in severe COVID-19
NOS inhibition reverses TLR2-induced chondrocyte dysfunction and attenuates age-related osteoarthritis
Cross-regulation of antibody responses against the SARS-CoV-2 Spike protein and commensal microbiota via molecular mimicry
MicroRNA-223 Dampens Pulmonary Inflammation during Pneumococcal Pneumonia
Resident memory CD4 <sup>+</sup> T lymphocytes mobilize from bone marrow to contribute to a systemic secondary immune reaction
A pulmonologist's guide to perform and analyse cross-species single lung cell transcriptomics
Optimization of chondrocyte isolation from human articular cartilage to preserve the chondrocyte transcriptome
Untimely TGFβ responses in COVID-19 limit antiviral functions of NK cells
SARS-CoV-2 in severe COVID-19 induces a TGF-β-dominated chronic immune response that does not target itself
Combining segmental bulk- and single-cell RNA-sequencing to define the chondrocyte gene expression signature in the murine knee joint
Targeting CD38 with Daratumumab in Refractory Systemic Lupus Erythematosus
Microbiota-Induced Type I Interferons Instruct a Poised Basal State of Dendritic Cells
Discrete populations of isotype-switched memory B lymphocytes are maintained in murine spleen and bone marrow
Group 3 Innate Lymphoid Cells Program a Distinct Subset of IL-22BP-Producing Dendritic Cells Demarcating Solitary Intestinal Lymphoid Tissues
Antigen‐driven PD‐1 <sup>+</sup> <i>TOX</i> <sup>+</sup> <i>BHLHE40</i> <sup>+</sup> and PD‐1 <sup>+</sup> <i>TOX</i> <sup>+</sup> <i>EOMES</i> <sup>+</sup> T lymphocytes regulate juvenile idiopathic arthritis <i>in situ</i>
Vitamin A controls the allergic response through T follicular helper cell as well as plasmablast differentiation
c-Maf-dependent Treg cell control of intestinal TH17 cells and IgA establishes host–microbiota homeostasis
Selective targeting of pro-inflammatory Th1 cells by microRNA-148a-specific antagomirs in vivo
Cleavage of roquin and regnase-1 by the paracaspase MALT1 releases their cooperatively repressed targets to promote TH17 differentiation
T cell activation induces proteasomal degradation of Argonaute and rapid remodeling of the microRNA repertoire
Eri1 degrades the stem-loop of oligouridylated histone mRNAs to induce replication-dependent decay