Tali Mazor

Molecular profiling of 888 pediatric tumors informs future precision trials and data-sharing initiatives in pediatric cancer

Analysis and Visualization of Longitudinal Genomic and Clinical Data from the AACR Project GENIE Biopharma Collaborative in cBioPortal

EPCO-39. CLARIFYING THE MOLECULAR CONSEQUENCES OF ONCOGENIC MUTATIONS THROUGH MULTISCALE AND MULTIOMIC ANALYSIS OF INDIVIDUAL TUMORS

AACR Project GENIE: 100,000 Cases and Beyond

Genomic analysis of early-stage lung cancer reveals a role for TP53 mutations in distant metastasis

OncoTree: A Cancer Classification System for Precision Oncology

Temozolomide-induced hypermutation is associated with distant recurrence and reduced survival after high-grade transformation of low-grade <i>IDH</i>-mutant gliomas

Human placental cytotrophoblast epigenome dynamics over gestation and alterations in placental disease

Longitudinal molecular trajectories of diffuse glioma in adults

Recurrent KBTBD4 small in-frame insertions and absence of DROSHA deletion or DICER1 mutation differentiate pineal parenchymal tumor of intermediate differentiation (PPTID) from pineoblastoma

Isocitrate dehydrogenase mutations suppress STAT1 and CD8+ T cell accumulation in gliomas

Clonal expansion and epigenetic reprogramming following deletion or amplification of mutant <i>IDH1</i>

Genome-wide DNA methylation is predictive of outcome in juvenile myelomonocytic leukemia

Intratumoral Heterogeneity of the Epigenome

Mutant IDH1 Expression Drives <i>TERT</i> Promoter Reactivation as Part of the Cellular Transformation Process

Somatic and Germline <i>TP53</i> Alterations in Second Malignant Neoplasms from Pediatric Cancer Survivors

The genomic landscape of juvenile myelomonocytic leukemia

DNA Methylation and Somatic Mutations Converge on the Cell Cycle and Define Similar Evolutionary Histories in Brain Tumors

Evolution of DNA repair defects during malignant progression of low-grade gliomas after temozolomide treatment

Mutational Analysis Reveals the Origin and Therapy-Driven Evolution of Recurrent Glioma

Direct Recruitment of Polycomb Repressive Complex 1 (PRC1) to Chromatin by Core Binding Transcription Factors

Direct Recruitment of Polycomb Repressive Complex 1 to Chromatin by Core Binding Transcription Factors