Area of research
Physiology · Cellular and Molecular Neuroscience
Research interest
Research interests include Chronic pain, Medicine, Microglia, Neuroscience, Spinal cord, and Psychology.
Recommendations for the inclusion and study of sex and gender in research
Single-cell RNA sequencing reveals time- and sex-specific responses of mouse spinal cord microglia to peripheral nerve injury and links ApoE to chronic pain
Long-term male-specific chronic pain via telomere- and p53‑mediated spinal cord cellular senescence
mTORC2 mediates structural plasticity in distal nociceptive endings that contributes to pain hypersensitivity following inflammation
The revised International Association for the Study of Pain definition of pain: concepts, challenges, and compromises
The development and use of facial grimace scales for pain measurement in animals
MicroRNA-19b predicts widespread pain and posttraumatic stress symptom risk in a sex-dependent manner following trauma exposure
Microglial P2X4R-evoked pain hypersensitivity is sexually dimorphic in rats
Comprehensive analysis of long noncoding RNA expression in dorsal root ganglion reveals cell-type specificity and dysregulation after nerve injury
Genome-wide association reveals contribution of MRAS to painful temporomandibular disorder in males
The MNK–eIF4E Signaling Axis Contributes to Injury-Induced Nociceptive Plasticity and the Development of Chronic Pain
Epiregulin and EGFR interactions are involved in pain processing
Inhibition of the kinase WNK1/HSN2 ameliorates neuropathic pain by restoring GABA inhibition
Different immune cells mediate mechanical pain hypersensitivity in male and female mice
Sex inclusion in basic research drives discovery
The nicotinic α6 subunit gene determines variability in chronic pain sensitivity via cross-inhibition of P2X2/3 receptors
Olfactory exposure to males, including men, causes stress and related analgesia in rodents
Quantifying Blood-Spinal Cord Barrier Permeability after Peripheral Nerve Injury in the Living Mouse
Constitutive μ-Opioid Receptor Activity Leads to Long-Term Endogenous Analgesia and Dependence
mTORC1 inhibition induces pain via IRS-1-dependent feedback activation of ERK
Genetically determined P2X7 receptor pore formation regulates variability in chronic pain sensitivity
Loss of Neuronal Potassium/Chloride Cotransporter 3 (KCC3) Is Responsible for the Degenerative Phenotype in a Conditional Mouse Model of Hereditary Motor and Sensory Neuropathy Associated with Agenesis of the Corpus Callosum