Area of research
Physiology · Cellular and Molecular Neuroscience
Research interest
Research interests include Sensitization, Medicine, Nociceptor, Neuroscience, Pharmacology, and AMPK.
eIF4E Phosphorylation Influences Bdnf mRNA Translation in Mouse Dorsal Root Ganglion Neurons
The MNK–eIF4E Signaling Axis Contributes to Injury-Induced Nociceptive Plasticity and the Development of Chronic Pain
Pharmacological activation of AMPK inhibits incision-evoked mechanical hypersensitivity and the development of hyperalgesic priming in mice
Neuroligin 2 regulates spinal GABAergic plasticity in hyperalgesic priming, a model of the transition from acute to chronic pain
The novel <scp>PAR</scp>2 ligand <scp>C</scp>391 blocks multiple <scp>PAR</scp>2 signalling pathways <i>in vitro</i> and <i>in vivo</i>
Meningeal norepinephrine produces headache behaviors in rats via actions both on dural afferents and fibroblasts
Local Translation and Retrograde Axonal Transport of CREB Regulates IL-6-Induced Nociceptive Plasticity
Inhibition of Carbonic Anhydrase Augments GABAA Receptor-Mediated Analgesia via a Spinal Mechanism of Action
mTORC1 inhibition induces pain via IRS-1-dependent feedback activation of ERK
BDNF Regulates Atypical PKC at Spinal Synapses to Initiate and Maintain a Centralized Chronic Pain State
Resveratrol Engages AMPK to Attenuate ERK and mTOR Signaling in Sensory Neurons and Inhibits Incision-Induced Acute and Chronic Pain